Overview
Sila Hopton practices in Newcastle Upon Tyne, United Kingdom. Ms. Hopton is rated as an Experienced expert by MediFind in the treatment of Myoclonic Epilepsy Associated With Ragged Red Fibers. Her top areas of expertise are Progressive External Ophthalmoplegia, Leigh Syndrome, Mitochondrial Complex 1 Deficiency, and Retinopathy Pigmentary Mental Retardation.
Her clinical research consists of co-authoring 25 peer reviewed articles. MediFind looks at clinical research from the past 15 years. In particular, she has co-authored 1 article in the study of Myoclonic Epilepsy Associated With Ragged Red Fibers.
Locations
Clinical Research
Clinical research consists of overseeing clinical studies of patients undergoing new treatments and therapies, and publishing articles in peer reviewed medical journals. Experts who actively participate in clinical research are generally at the forefront of the fields and aware of the most up-to-date advances in treatments for their patients.
Areas of Expertise
MediFind evaluates expertise by pulling from factors such as number of articles a doctor has published in medical journals, participation in clinical trials, speaking at industry conferences, prescribing and referral patterns, and strength of connections with other experts in their field.
Learn more about MediFind’s expert tiers
- Advanced
- Leigh SyndromeMs. Hopton isAdvanced. Learn about Leigh Syndrome.
- Mitochondrial Complex 1 Deficiency
- Progressive External Ophthalmoplegia
- Retinopathy Pigmentary Mental Retardation
- Experienced
- Acute Cerebellar AtaxiaMs. Hopton isExperienced. Learn about Acute Cerebellar Ataxia.
- Brachydactyly Mononen TypeMs. Hopton isExperienced. Learn about Brachydactyly Mononen Type.
- Brown SyndromeMs. Hopton isExperienced. Learn about Brown Syndrome.
- ChondrodystrophyMs. Hopton isExperienced. Learn about Chondrodystrophy.
- Collins Pope SyndromeMs. Hopton isExperienced. Learn about Collins Pope Syndrome.
- Cytochrome C Oxidase DeficiencyMs. Hopton isExperienced. Learn about Cytochrome C Oxidase Deficiency.