
Adam G. Rokita
Regensburg, BY, Germany
Last Updated: 09/26/2026
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Found 9 publications
Ca2+/calmodulin-dependent protein kinase II is essential in hyperacute pressure overload.
Journal: Journal of molecular and cellular cardiology
Published: August 04, 2019
Oxidized CaMKII and O-GlcNAcylation cause increased atrial fibrillation in diabetic mice by distinct mechanisms.
Journal: The Journal of clinical investigation
Published: June 16, 2017
Corrigendum: The mitochondrial uniporter controls fight or flight heart rate increases.
Journal: Nature communications
Published: June 04, 2015
The mitochondrial uniporter controls fight or flight heart rate increases.
Journal: Nature communications
Published: May 08, 2014
Oxidized Ca(2+)/calmodulin-dependent protein kinase II triggers atrial fibrillation.
Journal: Circulation
Published: September 14, 2013
Redox regulation of sodium and calcium handling.
Journal: Antioxidants & redox signaling
Published: August 21, 2012
Diabetes increases mortality after myocardial infarction by oxidizing CaMKII.
Journal: The Journal of clinical investigation
Published: June 11, 2012
Showing 1-9 of 9
Last Updated: 09/26/2026