Agata Kodron
Warsaw, MZ, Poland
Last Updated: 09/26/2026
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Found 10 publications
Immunoproteasome-specific Subunit Alterations as a Potential Therapeutic Target for Mitochondriopathies.
Journal: Journal of molecular biology
Published: February 24, 2025
Immunoproteasome-specific subunit PSMB9 induction is required to regulate cellular proteostasis upon mitochondrial dysfunction.
Journal: Nature communications
Published: February 08, 2022
The ubiquitin-proteasome system and its crosstalk with mitochondria as therapeutic targets in medicine.
Journal: Pharmacological research
Published: May 04, 2020
Testosterone increases apoptotic cell death and decreases mitophagy in Leber's hereditary optic neuropathy cells.
Journal: Journal of applied genetics
Published: July 21, 2019
Analysis of BNIP3 and BNIP3L/Nix expression in cybrid cell lines harboring two LHON-associated mutations.
Journal: Acta biochimica Polonica
Published: June 11, 2019
Mitochondrial DNA in pediatric leukemia patients.
Journal: Acta biochimica Polonica
Published: October 20, 2016
Investigating Leber's hereditary optic neuropathy: Cell models and future perspectives.
Journal: Mitochondrion
Published: March 04, 2016
Changing phenotypic expression in a patient with a mitochondrial encephalopathy due to 13042G>A de novo mutation--a 5 year follow up.
Journal: Metabolic brain disease
Published: September 30, 2014
m.3635G>A mutation as a cause of Leber hereditary optic neuropathy.
Journal: Journal of clinical pathology
Published: April 22, 2014
Mitochondrial encephalopathy in a patient with a 13042G>A de novo mutation.
Journal: Journal of clinical pathology
Published: August 14, 2012
Showing 1-10 of 10
Last Updated: 09/26/2026