Agnes Jamin - Publications

Recruitment of CXCR3+ T cells into injured tissues in adult IgA vasculitis patients correlates with disease activity.

Journal: Journal of autoimmunity

Published: November 07, 2018

Autoantibodies against podocytic UCHL1 are associated with idiopathic nephrotic syndrome relapses and induce proteinuria in mice.

Journal: Journal of autoimmunity

Published: October 11, 2017

New insights in the pathogenesis of immunoglobulin A vasculitis (Henoch-Schönlein purpura).

Journal: Autoimmunity reviews

Published: August 23, 2017

Biomarkers of IgA vasculitis nephritis in children.

Journal: PloS one

Published: June 23, 2017

Value of biomarkers for predicting immunoglobulin A vasculitis nephritis outcome in an adult prospective cohort.

Journal: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association

Published: April 20, 2017

Neonatal high protein intake enhances neonatal growth without significant adverse renal effects in spontaneous IUGR piglets.

Journal: Physiological reports

Published: April 13, 2017

Idiopathic nephrotic syndrome: the EBV hypothesis.

Journal: Pediatric research

Published: May 18, 2016

IgA1 Protease Treatment Reverses Mesangial Deposits and Hematuria in a Model of IgA Nephropathy.

Journal: Journal of the American Society of Nephrology : JASN

Published: August 04, 2015

Toll-like receptor 3 expression and function in childhood idiopathic nephrotic syndrome.

Journal: Clinical and experimental immunology

Published: May 22, 2015

A high-protein formula increases colonic peptide transporter 1 activity during neonatal life in low-birth-weight piglets and disturbs barrier function later in life.

Journal: The British journal of nutrition

Published: August 15, 2014

Dietary protein excess during neonatal life alters colonic microbiota and mucosal response to inflammatory mediators later in life in female pigs.

Journal: The Journal of nutrition

Published: June 14, 2013

Transglutaminase is essential for IgA nephropathy development acting through IgA receptors.

Journal: The Journal of experimental medicine

Published: March 28, 2012