Ali Bakr

Heidelberg, BW 69120, Germany

Last Updated: 09/26/2026

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Found 14 publications

APOBEC2 deficiency disrupts hematopoietic lineage commitment, resulting in emergence of dual identity lymphocytes in mice and humans.

APOBEC2 deficiency disrupts hematopoietic lineage commitment, resulting in emergence of dual identity lymphocytes in mice and humans.

Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: June 05, 2026

APOBEC2 deficiency disrupts hematopoietic lineage commitment, resulting in emergence of dual identity lymphocytes in mice and humans.

APOBEC2 deficiency disrupts hematopoietic lineage commitment, resulting in emergence of dual identity lymphocytes in mice and humans.

Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: June 05, 2026

ID3 deficiency alters chromatin accessibility at DSB sites and enhances vulnerability to HDAC inhibition.

ID3 deficiency alters chromatin accessibility at DSB sites and enhances vulnerability to HDAC inhibition.

Journal: International journal of cancer
Published: July 07, 2025

SRSF2 safeguards efficient transcription of DNA damage and repair genes.

SRSF2 safeguards efficient transcription of DNA damage and repair genes.

Journal: Cell reports
Published: February 01, 2024

ARID1A regulates DNA repair through chromatin organization and its deficiency triggers DNA damage-mediated anti-tumor immune response.

ARID1A regulates DNA repair through chromatin organization and its deficiency triggers DNA damage-mediated anti-tumor immune response.

Journal: Nucleic acids research
Published: December 08, 2023

Deposition of onco-histone H3.3-G34W leads to DNA repair deficiency and activates cGAS/STING-mediated immune responses.

Deposition of onco-histone H3.3-G34W leads to DNA repair deficiency and activates cGAS/STING-mediated immune responses.

Journal: International journal of cancer
Published: November 23, 2023

TAL1 activation in T-cell acute lymphoblastic leukemia: a novel oncogenic 3' neo-enhancer.

TAL1 activation in T-cell acute lymphoblastic leukemia: a novel oncogenic 3' neo-enhancer.

Journal: Haematologica
Published: June 16, 2022

Selective inhibitors of bromodomain BD1 and BD2 of BET proteins modulate radiation-induced profibrotic fibroblast responses.

Selective inhibitors of bromodomain BD1 and BD2 of BET proteins modulate radiation-induced profibrotic fibroblast responses.

Journal: International journal of cancer
Published: October 29, 2021

ID3 promotes homologous recombination via non-transcriptional and transcriptional mechanisms and its loss confers sensitivity to PARP inhibition.

ID3 promotes homologous recombination via non-transcriptional and transcriptional mechanisms and its loss confers sensitivity to PARP inhibition.

Journal: Nucleic acids research
Published: July 19, 2021

Epigenetic Modulation of Radiation-Induced Diacylglycerol Kinase Alpha Expression Prevents Pro-Fibrotic Fibroblast Response.

Epigenetic Modulation of Radiation-Induced Diacylglycerol Kinase Alpha Expression Prevents Pro-Fibrotic Fibroblast Response.

Journal: Cancers
Published: March 08, 2021

Identification of therapeutic targets of the hijacked super-enhancer complex in EVI1-rearranged leukemia.

Identification of therapeutic targets of the hijacked super-enhancer complex in EVI1-rearranged leukemia.

Journal: Leukemia
Published: July 02, 2020

Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1.

Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1.

Journal: Oncotarget
Published: July 07, 2016
Showing 1-12 of 14

Last Updated: 09/26/2026