Alvaro Macias

Madrid, MD, Spain

Last Updated: 09/26/2026

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Found 26 publications

MOG1L18F-mediated increase in late sodium current produces Long QT Syndrome.

MOG1L18F-mediated increase in late sodium current produces Long QT Syndrome.

Journal: medRxiv : the preprint server for health sciences
Published: December 25, 2025

Ventricular fibrillation dynamics reveal regional asymmetry in resilience to cardiac arrest and predict clinical outcome.

Ventricular fibrillation dynamics reveal regional asymmetry in resilience to cardiac arrest and predict clinical outcome.

Journal: Cardiovascular research
Published: December 04, 2025

Repurposing polyamines to prevent life-threatening arrhythmias in Short QT Syndrome type 3.

Repurposing polyamines to prevent life-threatening arrhythmias in Short QT Syndrome type 3.

Journal: Nature communications
Published: November 27, 2025

Lgi3-4 proteins modulate the KV1.5 channelosome and are potential therapeutic targets for atrial fibrillation.

Lgi3-4 proteins modulate the KV1.5 channelosome and are potential therapeutic targets for atrial fibrillation.

Journal: Cardiovascular research
Published: June 24, 2025

Kir2.1 mutations differentially increase the risk of flecainide proarrhythmia in Andersen Tawil Syndrome.

Kir2.1 mutations differentially increase the risk of flecainide proarrhythmia in Andersen Tawil Syndrome.

Journal: medRxiv : the preprint server for health sciences
Published: December 23, 2024

Extracellular Kir2.1C122Y Mutant Upsets Kir2.1-PIP2 Bonds and Is Arrhythmogenic in Andersen-Tawil Syndrome.

Extracellular Kir2.1C122Y Mutant Upsets Kir2.1-PIP2 Bonds and Is Arrhythmogenic in Andersen-Tawil Syndrome.

Journal: Circulation research
Published: March 18, 2024

MCJ: A mitochondrial target for cardiac intervention in pulmonary hypertension.

MCJ: A mitochondrial target for cardiac intervention in pulmonary hypertension.

Journal: Science advances
Published: January 19, 2024

The Kir2.1E299V mutation increases atrial fibrillation vulnerability while protecting the ventricles against arrhythmias in a mouse model of short QT syndrome type 3.

The Kir2.1E299V mutation increases atrial fibrillation vulnerability while protecting the ventricles against arrhythmias in a mouse model of short QT syndrome type 3.

Journal: Cardiovascular research
Published: July 04, 2023

Extracellular cysteine disulfide bond break at Cys122 disrupts PIP2-dependent Kir2.1 channel function and leads to arrhythmias in Andersen-Tawil Syndrome.

Extracellular cysteine disulfide bond break at Cys122 disrupts PIP2-dependent Kir2.1 channel function and leads to arrhythmias in Andersen-Tawil Syndrome.

Journal: bioRxiv : the preprint server for biology
Published: June 19, 2023

Coronary and carotid artery dysfunction and KV7 overexpression in a mouse model of Hutchinson-Gilford progeria syndrome.

Coronary and carotid artery dysfunction and KV7 overexpression in a mouse model of Hutchinson-Gilford progeria syndrome.

Journal: GeroScience
Published: March 30, 2023

Kir2.1 dysfunction at the sarcolemma and the sarcoplasmic reticulum causes arrhythmias in a mouse model of Andersen-Tawil syndrome type 1.

Kir2.1 dysfunction at the sarcolemma and the sarcoplasmic reticulum causes arrhythmias in a mouse model of Andersen-Tawil syndrome type 1.

Journal: Nature cardiovascular research
Published: March 28, 2022

Molecular stratification of arrhythmogenic mechanisms in the Andersen Tawil syndrome.

Molecular stratification of arrhythmogenic mechanisms in the Andersen Tawil syndrome.

Journal: Cardiovascular research
Published: March 18, 2022
Showing 1-12 of 26

Last Updated: 09/26/2026