Amanda Penco

Genoa, Italy

Last Updated: 09/26/2026

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Found 15 publications

Full-length TDP-43 and its C-terminal domain form filaments in vitro having non-amyloid properties.

Full-length TDP-43 and its C-terminal domain form filaments in vitro having non-amyloid properties.

Journal: Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis
Published: October 07, 2020

Epigallocatechin-3-gallate and related phenol compounds redirect the amyloidogenic aggregation pathway of ataxin-3 towards non-toxic aggregates and prevent toxicity in neural cells and Caenorhabditis elegans animal model.

Epigallocatechin-3-gallate and related phenol compounds redirect the amyloidogenic aggregation pathway of ataxin-3 towards non-toxic aggregates and prevent toxicity in neural cells and Caenorhabditis elegans animal model.

Journal: Human molecular genetics
Published: April 03, 2017

Biochemical and Electrophysiological Modification of Amyloid Transthyretin on Cardiomyocytes.

Biochemical and Electrophysiological Modification of Amyloid Transthyretin on Cardiomyocytes.

Journal: Biophysical journal
Published: April 12, 2016

Effect of molecular chaperones on aberrant protein oligomers in vitro: super-versus sub-stoichiometric chaperone concentrations.

Effect of molecular chaperones on aberrant protein oligomers in vitro: super-versus sub-stoichiometric chaperone concentrations.

Journal: Biological chemistry
Published: September 10, 2015

Protein conformational perturbations in hereditary amyloidosis: Differential impact of single point mutations in ApoAI amyloidogenic variants.

Protein conformational perturbations in hereditary amyloidosis: Differential impact of single point mutations in ApoAI amyloidogenic variants.

Journal: Biochimica et biophysica acta
Published: July 17, 2015

A covalent homodimer probing early oligomers along amyloid aggregation.

A covalent homodimer probing early oligomers along amyloid aggregation.

Journal: Scientific reports
Published: June 08, 2015

The H50Q mutation induces a 10-fold decrease in the solubility of α-synuclein.

The H50Q mutation induces a 10-fold decrease in the solubility of α-synuclein.

Journal: The Journal of biological chemistry
Published: December 16, 2014

Wild type beta-2 microglobulin and DE loop mutants display a common fibrillar architecture.

Wild type beta-2 microglobulin and DE loop mutants display a common fibrillar architecture.

Journal: PloS one
Published: October 17, 2014

Epigallocatechin-3-gallate and tetracycline differently affect ataxin-3 fibrillogenesis and reduce toxicity in spinocerebellar ataxia type 3 model.

Epigallocatechin-3-gallate and tetracycline differently affect ataxin-3 fibrillogenesis and reduce toxicity in spinocerebellar ataxia type 3 model.

Journal: Human molecular genetics
Published: July 18, 2014

TDP-43 inclusion bodies formed in bacteria are structurally amorphous, non-amyloid and inherently toxic to neuroblastoma cells.

TDP-43 inclusion bodies formed in bacteria are structurally amorphous, non-amyloid and inherently toxic to neuroblastoma cells.

Journal: PloS one
Published: October 17, 2013

Structure, folding dynamics, and amyloidogenesis of D76N β2-microglobulin: roles of shear flow, hydrophobic surfaces, and α-crystallin.

Structure, folding dynamics, and amyloidogenesis of D76N β2-microglobulin: roles of shear flow, hydrophobic surfaces, and α-crystallin.

Journal: The Journal of biological chemistry
Published: September 10, 2013

Different ataxin-3 amyloid aggregates induce intracellular Ca(2+) deregulation by different mechanisms in cerebellar granule cells.

Different ataxin-3 amyloid aggregates induce intracellular Ca(2+) deregulation by different mechanisms in cerebellar granule cells.

Journal: Biochimica et biophysica acta
Published: April 15, 2013
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Last Updated: 09/26/2026