Ambreen Rehman
London, ENG E12AT, United Kingdom
Last Updated: 09/26/2026
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Found 8 publications
YAP Inhibition by Verteporfin Causes Downregulation of Desmosomal Genes and Proteins Leading to the Disintegration of Intercellular Junctions.
Journal: Life (Basel, Switzerland)
Published: March 25, 2022
Evolving Mechanisms in the Pathophysiology of Pemphigus Vulgaris: A Review Emphasizing the Role of Desmoglein 3 in Regulating p53 and the Yes-Associated Protein.
Journal: Life (Basel, Switzerland)
Published: June 08, 2021
Gingival Crevicular Fluid (GCF): A Diagnostic Tool for the Detection of Periodontal Health and Diseases.
Journal: Molecules (Basel, Switzerland)
Published: February 06, 2021
Oxidative Stress-Mediated YAP Dysregulation Contributes to the Pathogenesis of Pemphigus Vulgaris.
Journal: Frontiers in immunology
Published: January 04, 2021
Evidence for the Desmosomal Cadherin Desmoglein-3 in Regulating YAP and Phospho-YAP in Keratinocyte Responses to Mechanical Forces.
Journal: International journal of molecular sciences
Published: November 06, 2019
The desmosomal cadherin desmoglein-3 acts as a keratinocyte anti-stress protein via suppression of p53.
Journal: Cell death & disease
Published: June 13, 2019
Desmoglein-3 acts as a pro-survival protein by suppressing reactive oxygen species and doming whilst augmenting the tight junctions in MDCK cells.
Journal: Mechanisms of ageing and development
Published: March 14, 2019
Areca nut alkaloids induce irreparable DNA damage and senescence in fibroblasts and may create a favourable environment for tumour progression.
Journal: Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
Published: August 17, 2015
Showing 1-8 of 8
Last Updated: 09/26/2026