Anandhkumar Raju

61 Biopolis Drive, Proteos
Singapore, 138673, Singapore

Last Updated: 09/26/2026

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Found 21 publications

A genome-wide genetic screen reveals the P2Y2-integrin axis as a stabilizer of EGFR mutants in non-small cell lung cancer (NSCLC).

A genome-wide genetic screen reveals the P2Y2-integrin axis as a stabilizer of EGFR mutants in non-small cell lung cancer (NSCLC).

Journal: Science advances

Published: January 21, 2026

Retraction Note: Tumour microenvironment programming by an RNA-RNA-binding protein complex creates a druggable vulnerability in IDH-wild-type glioblastoma.

Retraction Note: Tumour microenvironment programming by an RNA-RNA-binding protein complex creates a druggable vulnerability in IDH-wild-type glioblastoma.

Journal: Nature cell biology

Published: January 17, 2025

The transcription complex p52-ETS1 is essential for germinal center formation.

The transcription complex p52-ETS1 is essential for germinal center formation.

Journal: Nature immunology

Published: August 15, 2024

Iron-(Fe3+)-Dependent Reactivation of Telomerase Drives Colorectal Cancers.

Iron-(Fe3+)-Dependent Reactivation of Telomerase Drives Colorectal Cancers.

Journal: Cancer discovery

Published: November 17, 2023

Tumour microenvironment programming by an RNA-RNA-binding protein complex creates a druggable vulnerability in IDH-wild-type glioblastoma.

Tumour microenvironment programming by an RNA-RNA-binding protein complex creates a druggable vulnerability in IDH-wild-type glioblastoma.

Journal: Nature cell biology

Published: March 21, 2023

Super-silencer perturbation by EZH2 and REST inhibition leads to large loss of chromatin interactions and reduction in cancer growth.

Super-silencer perturbation by EZH2 and REST inhibition leads to large loss of chromatin interactions and reduction in cancer growth.

Journal: Nature structural & molecular biology

Published: April 03, 2022

Dependency of NELF-E-SLUG-KAT2B epigenetic axis in breast cancer carcinogenesis.

Dependency of NELF-E-SLUG-KAT2B epigenetic axis in breast cancer carcinogenesis.

Journal: Nature communications

Published: March 04, 2022

p53-NEIL1 co-abnormalities induce genomic instability and promote synthetic lethality with Chk1 inhibition in multiple myeloma having concomitant 17p13(del) and 1q21(gain).

p53-NEIL1 co-abnormalities induce genomic instability and promote synthetic lethality with Chk1 inhibition in multiple myeloma having concomitant 17p13(del) and 1q21(gain).

Journal: Oncogene

Published: August 30, 2021

GREB1: An evolutionarily conserved protein with a glycosyltransferase domain links ERα glycosylation and stability to cancer.

GREB1: An evolutionarily conserved protein with a glycosyltransferase domain links ERα glycosylation and stability to cancer.

Journal: Science advances

Published: August 09, 2020

Targeting N-myristoylation for therapy of B-cell lymphomas.

Targeting N-myristoylation for therapy of B-cell lymphomas.

Journal: Nature communications

Published: November 18, 2019

H3K27me3-rich genomic regions can function as silencers to repress gene expression via chromatin interactions.

H3K27me3-rich genomic regions can function as silencers to repress gene expression via chromatin interactions.

Journal: Nature communications

Published: July 03, 2019

A Role of Agrin in Maintaining the Stability of Vascular Endothelial Growth Factor Receptor-2 during Tumor Angiogenesis.

A Role of Agrin in Maintaining the Stability of Vascular Endothelial Growth Factor Receptor-2 during Tumor Angiogenesis.

Journal: Cell reports

Published: August 08, 2018

Showing 1-12 of 21

Last Updated: 09/26/2026