Anders Nordholm

Reply to: high prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.

Journal: Osteoporosis international : a journal established as result of cooperation between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA

Published: June 22, 2026

High prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.

Journal: Osteoporosis international : a journal established as result of cooperation between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA

Published: November 28, 2025

Plasma activin A rises with declining kidney function and is independently associated with mortality in patients with chronic kidney disease.

Journal: Clinical kidney journal

Published: June 21, 2023

Roles of NAD+ in Acute and Chronic Kidney Diseases.

Journal: International journal of molecular sciences

Published: October 27, 2022

The Calcified Vasculature in Chronic Kidney Disease Secretes Factors that Inhibit Bone Mineralization.

Journal: JBMR plus

Published: November 09, 2021

Hypomorphic expression of parathyroid Bmal1 disrupts the internal parathyroid circadian clock and increases parathyroid cell proliferation in response to uremia.

Journal: Kidney international

Published: September 15, 2021

Diurnal variation of magnesium and the mineral metabolism in patients with chronic kidney disease.

Journal: Bone reports

Published: July 15, 2021

Effect of NAD+ boosting on kidney ischemia-reperfusion injury.

Journal: PloS one

Published: February 15, 2021

Chronic Kidney Disease-Induced Vascular Calcification Impairs Bone Metabolism.

Journal: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research

Published: May 15, 2020

A molecular circadian clock operates in the parathyroid gland and is disturbed in chronic kidney disease associated bone and mineral disorder.

Journal: Kidney international

Published: January 05, 2020

Reply to: high prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.

Reply to: high prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.

High prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.

High prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.

Plasma activin A rises with declining kidney function and is independently associated with mortality in patients with chronic kidney disease.

Plasma activin A rises with declining kidney function and is independently associated with mortality in patients with chronic kidney disease.

Roles of NAD+ in Acute and Chronic Kidney Diseases.

Roles of NAD+ in Acute and Chronic Kidney Diseases.

The Calcified Vasculature in Chronic Kidney Disease Secretes Factors that Inhibit Bone Mineralization.

The Calcified Vasculature in Chronic Kidney Disease Secretes Factors that Inhibit Bone Mineralization.

Hypomorphic expression of parathyroid Bmal1 disrupts the internal parathyroid circadian clock and increases parathyroid cell proliferation in response to uremia.

Hypomorphic expression of parathyroid Bmal1 disrupts the internal parathyroid circadian clock and increases parathyroid cell proliferation in response to uremia.

Diurnal variation of magnesium and the mineral metabolism in patients with chronic kidney disease.

Diurnal variation of magnesium and the mineral metabolism in patients with chronic kidney disease.

Effect of NAD+ boosting on kidney ischemia-reperfusion injury.

Effect of NAD+ boosting on kidney ischemia-reperfusion injury.

Chronic Kidney Disease-Induced Vascular Calcification Impairs Bone Metabolism.

Chronic Kidney Disease-Induced Vascular Calcification Impairs Bone Metabolism.

A molecular circadian clock operates in the parathyroid gland and is disturbed in chronic kidney disease associated bone and mineral disorder.

A molecular circadian clock operates in the parathyroid gland and is disturbed in chronic kidney disease associated bone and mineral disorder.

Circadian rhythm of activin A and related parameters of mineral metabolism in normal and uremic rats.

Circadian rhythm of activin A and related parameters of mineral metabolism in normal and uremic rats.

Effect of inhibition of CBP-coactivated β-catenin-mediated Wnt signalling in uremic rats with vascular calcifications.

Effect of inhibition of CBP-coactivated β-catenin-mediated Wnt signalling in uremic rats with vascular calcifications.