Andrea Toth

Debrecen, HB 4032, Hungary

Last Updated: 09/26/2026

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Found 23 publications

Iron in Vascular Calcification: Pro-Calcific Agent or Protective Modulator?

Iron in Vascular Calcification: Pro-Calcific Agent or Protective Modulator?

Journal: International journal of molecular sciences

Published: September 10, 2025

The OsteoSense Imaging Agent Identifies Organ-Specific Patterns of Soft Tissue Calcification in an Adenine-Induced Chronic Kidney Disease Mouse Model.

The OsteoSense Imaging Agent Identifies Organ-Specific Patterns of Soft Tissue Calcification in an Adenine-Induced Chronic Kidney Disease Mouse Model.

Journal: International journal of molecular sciences

Published: July 17, 2025

Hypoxia-Induced Changes in Endothelial Cell Phenotype and Function.

Hypoxia-Induced Changes in Endothelial Cell Phenotype and Function.

Journal: Antioxidants & redox signaling

Published: May 28, 2025

Zinc Ameliorates High Pi and Ca-Mediated Osteogenic Differentiation of Mesenchymal Stem Cells.

Zinc Ameliorates High Pi and Ca-Mediated Osteogenic Differentiation of Mesenchymal Stem Cells.

Journal: Nutrients

Published: October 23, 2024

In Vitro Models of Cardiovascular Calcification.

In Vitro Models of Cardiovascular Calcification.

Journal: Biomedicines

Published: August 30, 2024

Activation of PERK/eIF2α/ATF4/CHOP branch of endoplasmic reticulum stress response and cooperation between HIF-1α and ATF4 promotes Daprodustat-induced vascular calcification.

Activation of PERK/eIF2α/ATF4/CHOP branch of endoplasmic reticulum stress response and cooperation between HIF-1α and ATF4 promotes Daprodustat-induced vascular calcification.

Journal: Frontiers in pharmacology

Published: March 11, 2024

High phosphate and calcium induce osteoblastic phenotype switching and calcification of corneal epithelial cells in a Runx2-dependent and synergistic manner; a possible mechanism of chronic kidney disease-associated corneal calcification.

High phosphate and calcium induce osteoblastic phenotype switching and calcification of corneal epithelial cells in a Runx2-dependent and synergistic manner; a possible mechanism of chronic kidney disease-associated corneal calcification.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: January 10, 2024

High glucose promotes osteogenic differentiation of human lens epithelial cells through hypoxia-inducible factor (HIF) activation.

High glucose promotes osteogenic differentiation of human lens epithelial cells through hypoxia-inducible factor (HIF) activation.

Journal: Journal of cellular physiology

Published: August 04, 2023

Hypoxia-inducible factor activation promotes osteogenic transition of valve interstitial cells and accelerates aortic valve calcification in a mice model of chronic kidney disease.

Hypoxia-inducible factor activation promotes osteogenic transition of valve interstitial cells and accelerates aortic valve calcification in a mice model of chronic kidney disease.

Journal: Frontiers in cardiovascular medicine

Published: February 17, 2023

The mechanosensitive Piezo1 channels contribute to the arterial medial calcification.

The mechanosensitive Piezo1 channels contribute to the arterial medial calcification.

Journal: Frontiers in physiology

Published: September 05, 2022

Activation of Nrf2/HO-1 Antioxidant Pathway by Heme Attenuates Calcification of Human Lens Epithelial Cells.

Activation of Nrf2/HO-1 Antioxidant Pathway by Heme Attenuates Calcification of Human Lens Epithelial Cells.

Journal: Pharmaceuticals (Basel, Switzerland)

Published: March 08, 2022

Daprodustat Accelerates High Phosphate-Induced Calcification Through the Activation of HIF-1 Signaling.

Daprodustat Accelerates High Phosphate-Induced Calcification Through the Activation of HIF-1 Signaling.

Journal: Frontiers in pharmacology

Published: October 19, 2021

Showing 1-12 of 23

Last Updated: 09/26/2026