Anfeng Mu
Kyoto, Japan
Last Updated: 09/26/2026
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Found 17 publications
SLFN11 inhibition rescues the Fanconi anemia phenotype by stabilizing stalled replication forks
Journal: [Rinsho ketsueki] The Japanese journal of clinical hematology
Published: January 01, 2024
The ribonuclease domain function is dispensable for SLFN11 to mediate cell fate decision during replication stress response.
Journal: Genes to cells : devoted to molecular & cellular mechanisms
Published: June 01, 2023
Fanconi anemia and Aldehyde Degradation Deficiency Syndrome: Metabolism and DNA repair protect the genome and hematopoiesis from endogenous DNA damage.
Journal: DNA repair
Published: May 12, 2023
Effects of the major formaldehyde catalyzer ADH5 on phenotypes of fanconi anemia zebrafish model.
Journal: Molecular biology reports
Published: March 23, 2023
Mouse Slfn8 and Slfn9 genes complement human cells lacking SLFN11 during the replication stress response.
Journal: Communications biology
Published: February 15, 2023
Aldehyde degradation deficiency (ADD) syndrome: discovery of a novel fanconi anemia-like inherited BMF syndrome due to combined ADH5/ALDH2 deficiency
Journal: [Rinsho ketsueki] The Japanese journal of clinical hematology
Published: July 05, 2021
SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells.
Journal: Blood
Published: October 18, 2019
Enhancements of the production of bilirubin and the expression of β-globin by carbon monoxide during erythroid differentiation.
Journal: FEBS letters
Published: February 25, 2016
Last Updated: 09/26/2026