Angela Poehlmann-Nitsche

Magdeburg, ST, Germany

Last Updated: 09/26/2026

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Found 12 publications

Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide-Associated Ulcerative Colitis through JNK Inactivation and Chromatin Binding.

Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide-Associated Ulcerative Colitis through JNK Inactivation and Chromatin Binding.

Journal: Oxidative medicine and cellular longevity

Published: December 20, 2016

Inactivation of JNK2 as carcinogenic factor in colitis-associated and sporadic colorectal carcinogenesis.

Inactivation of JNK2 as carcinogenic factor in colitis-associated and sporadic colorectal carcinogenesis.

Journal: Carcinogenesis

Published: August 08, 2016

DUOX2 and DUOXA2 form the predominant enzyme system capable of producing the reactive oxygen species H2O2 in active ulcerative colitis and are modulated by 5-aminosalicylic acid.

DUOX2 and DUOXA2 form the predominant enzyme system capable of producing the reactive oxygen species H2O2 in active ulcerative colitis and are modulated by 5-aminosalicylic acid.

Journal: Inflammatory bowel diseases

Published: February 05, 2014

Repeated H2 O2 exposure drives cell cycle progression in an in vitro model of ulcerative colitis.

Repeated H2 O2 exposure drives cell cycle progression in an in vitro model of ulcerative colitis.

Journal: Journal of cellular and molecular medicine

Published: July 18, 2013

ATF2 knockdown reinforces oxidative stress-induced apoptosis in TE7 cancer cells.

ATF2 knockdown reinforces oxidative stress-induced apoptosis in TE7 cancer cells.

Journal: Journal of cellular and molecular medicine

Published: October 27, 2012

Non-apoptotic function of caspases in a cellular model of hydrogen peroxide-associated colitis.

Non-apoptotic function of caspases in a cellular model of hydrogen peroxide-associated colitis.

Journal: Journal of cellular and molecular medicine

Published: August 03, 2012

Inflammation and Barrett's carcinogenesis.

Inflammation and Barrett's carcinogenesis.

Journal: Pathology, research and practice

Published: May 01, 2012

Cutting edge: Chk1 directs senescence and mitotic catastrophe in recovery from G₂ checkpoint arrest.

Cutting edge: Chk1 directs senescence and mitotic catastrophe in recovery from G₂ checkpoint arrest.

Journal: Journal of cellular and molecular medicine

Published: August 19, 2010

Importance of DNA damage checkpoints in the pathogenesis of human cancers.

Importance of DNA damage checkpoints in the pathogenesis of human cancers.

Journal: Pathology, research and practice

Published: August 03, 2010

RETRACTED: Identification of phosphorylated p38 as a novel DAPK-interacting partner during TNFalpha-induced apoptosis in colorectal tumor cells.

RETRACTED: Identification of phosphorylated p38 as a novel DAPK-interacting partner during TNFalpha-induced apoptosis in colorectal tumor cells.

Journal: The American journal of pathology

Published: July 25, 2009

Trichostatin A causes p53 to switch oxidative-damaged colorectal cancer cells from cell cycle arrest into apoptosis.

Trichostatin A causes p53 to switch oxidative-damaged colorectal cancer cells from cell cycle arrest into apoptosis.

Journal: Journal of cellular and molecular medicine

Published: April 19, 2008

K-ras mutation detection in colorectal cancer using the Pyrosequencing technique.

K-ras mutation detection in colorectal cancer using the Pyrosequencing technique.

Journal: Pathology, research and practice

Published: April 11, 2007

Showing 1-12 of 12

Last Updated: 09/26/2026