Anita Aperia
Solna, AB 17121, Sweden
Last Updated: 09/26/2026
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Found 32 publications
ATP1A3 dysfunction causes motor hyperexcitability and afterhyperpolarization loss in a dystonia model.
Journal: Brain : a journal of neurology
Published: May 17, 2024
A missense mutation converts the Na+,K+-ATPase into an ion channel and causes therapy-resistant epilepsy.
Journal: The Journal of biological chemistry
Published: May 04, 2021
An interaction between PRRT2 and Na+/K+ ATPase contributes to the control of neuronal excitability.
Journal: Cell death & disease
Published: October 26, 2020
Super-resolution microscopy reveals that Na+/K+-ATPase signaling protects against glucose-induced apoptosis by deactivating Bad.
Journal: Cell death & disease
Published: October 18, 2020
Mending Fences: Na,K-ATPase signaling via Ca2+ in the maintenance of epithelium integrity.
Journal: Cell calcium
Published: February 12, 2020
Ouabain Modulates the Functional Interaction Between Na,K-ATPase and NMDA Receptor.
Journal: Molecular neurobiology
Published: January 27, 2020
Prompt apoptotic response to high glucose in SGLT-expressing renal cells.
Journal: American journal of physiology. Renal physiology
Published: March 14, 2019
Regulation of Neuronal Na,K-ATPase by Extracellular Scaffolding Proteins.
Journal: International journal of molecular sciences
Published: July 02, 2018
AT1-receptor response to non-saturating Ang-II concentrations is amplified by calcium channel blockers.
Journal: BMC cardiovascular disorders
Published: December 19, 2016
Na+-K+-ATPase, a new class of plasma membrane receptors.
Journal: American journal of physiology. Cell physiology
Published: January 22, 2016
p11 regulates the surface localization of mGluR5.
Journal: Molecular psychiatry
Published: November 19, 2015
Last Updated: 09/26/2026