Anna-lena Forst

Regensburg, BY 93053, Germany

Last Updated: 09/26/2026

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Found 16 publications

Inhibition of (interstitial) P2Y6 receptors attenuates fibrosis progression.

Inhibition of (interstitial) P2Y6 receptors attenuates fibrosis progression.

Journal: Research square

Published: April 10, 2026

Inhibition of (interstitial) P2Y6 receptors attenuates fibrosis progression.

Inhibition of (interstitial) P2Y6 receptors attenuates fibrosis progression.

Journal: Pflugers Archiv : European journal of physiology

Published: March 18, 2026

Inhibition of (interstitial) P2Y6 receptors attenuates renal fibrosis progression.

Inhibition of (interstitial) P2Y6 receptors attenuates renal fibrosis progression.

Journal: bioRxiv : the preprint server for biology

Published: February 09, 2026

The endocrine product of renal (preglomerular) contractile pericytes depends on prolyl-4-hydroxylases 2 and 3.

The endocrine product of renal (preglomerular) contractile pericytes depends on prolyl-4-hydroxylases 2 and 3.

Journal: The Journal of physiology

Published: December 06, 2025

Hypoxia-inducible factor-2 stabilization is not sufficient to induce erythropoietin production in deeper medullary fibroblasts.

Hypoxia-inducible factor-2 stabilization is not sufficient to induce erythropoietin production in deeper medullary fibroblasts.

Journal: The Journal of physiology

Published: February 26, 2025

EAST/SeSAME Syndrome and Beyond: The Spectrum of Kir4.1- and Kir5.1-Associated Channelopathies.

EAST/SeSAME Syndrome and Beyond: The Spectrum of Kir4.1- and Kir5.1-Associated Channelopathies.

Journal: Frontiers in physiology

Published: January 11, 2022

A Founder Mutation in EHD1 Presents with Tubular Proteinuria and Deafness.

A Founder Mutation in EHD1 Presents with Tubular Proteinuria and Deafness.

Journal: Journal of the American Society of Nephrology : JASN

Published: October 06, 2021

Distinct Mitochondrial Pathologies Caused by Mutations of the Proximal Tubular Enzymes EHHADH and GATM.

Distinct Mitochondrial Pathologies Caused by Mutations of the Proximal Tubular Enzymes EHHADH and GATM.

Journal: Frontiers in physiology

Published: May 27, 2021

Genetic variants in eleven central and peripheral chemoreceptor genes in sudden infant death syndrome.

Genetic variants in eleven central and peripheral chemoreceptor genes in sudden infant death syndrome.

Journal: Pediatric research

Published: February 05, 2021

Defects in KCNJ16 Cause a Novel Tubulopathy with Hypokalemia, Salt Wasting, Disturbed Acid-Base Homeostasis, and Sensorineural Deafness.

Defects in KCNJ16 Cause a Novel Tubulopathy with Hypokalemia, Salt Wasting, Disturbed Acid-Base Homeostasis, and Sensorineural Deafness.

Journal: Journal of the American Society of Nephrology : JASN

Published: November 11, 2020

Novel mutations in the KCNJ10 gene associated to a distinctive ataxia, sensorineural hearing loss and spasticity clinical phenotype.

Novel mutations in the KCNJ10 gene associated to a distinctive ataxia, sensorineural hearing loss and spasticity clinical phenotype.

Journal: Neurogenetics

Published: August 06, 2019

Collecting system-specific deletion of Kcnj10 predisposes for thiazide- and low-potassium diet-induced hypokalemia.

Collecting system-specific deletion of Kcnj10 predisposes for thiazide- and low-potassium diet-induced hypokalemia.

Journal: Kidney international

Published: April 19, 2019

Showing 1-12 of 16

Last Updated: 09/26/2026