Anna Moles
Barcelona, CT, Spain
Last Updated: 09/26/2026
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Found 28 publications
Corrigendum to: "A TLR2/S100A9/CXCL-2 signaling network is necessary for neutrophil recruitment in acute and chronic liver injury in the mouse" [J Hepatol (2014) 60: 782-91].
Journal: Journal of hepatology
Published: July 06, 2026
VPS35 controls hepatocellular proliferation through SRC signalling and promotes diethyl nitrosamine-induced tumor initiation.
Journal: Cellular and molecular gastroenterology and hepatology
Published: September 20, 2025
Vacuolar Protein Sorting 35 Controls Hepatocellular Proliferation Through SRC Signaling and Promotes Diethyl Nitrosamine-Induced Tumor Initiation.
Journal: Cellular and molecular gastroenterology and hepatology
Published: September 20, 2025
Hepatic Stellate Cells Functional Heterogeneity in Liver Cancer.
Journal: Seminars in liver disease
Published: March 05, 2025
Patient-derived liver organoids recapitulate liver epithelial heterogeneity and enable precision modeling of alcohol-related liver disease.
Journal: Journal of hepatology
Published: September 10, 2024
Reprogramming macrophages to treat liver diseases.
Journal: Hepatology (Baltimore, Md.)
Published: July 17, 2024
Cathepsin D is essential for the degradomic shift of macrophages required to resolve liver fibrosis.
Cathepsin D is essential for the degradomic shift of macrophages required to resolve liver fibrosis.
Journal: Molecular metabolism
Published: May 14, 2024
Trajectory analysis of hepatic stellate cell differentiation reveals metabolic regulation of cell commitment and fibrosis.
Journal: Nature communications
Published: January 05, 2024
Ductular reaction-associated neutrophils promote biliary epithelium proliferation in chronic liver disease.
Journal: Journal of hepatology
Published: December 02, 2022
The multifaceted role of cathepsins in liver disease.
Journal: Journal of hepatology
Published: March 29, 2021
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
Journal: Autophagy
Published: February 26, 2021
NK cells eliminate Epstein-Barr virus bound to B cells through a specific antibody-mediated uptake.
Journal: PLoS pathogens
Published: October 04, 2020
Last Updated: 09/26/2026