Anna Vilalta
Cambridge, ENG, United Kingdom
Last Updated: 09/26/2026
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Found 17 publications
Correction to: Galectin-3, a novel endogenous TREM2 ligand, detrimentally regulates inflammatory response in Alzheimer's disease.
Journal: Acta neuropathologica
Published: February 15, 2023
Inflammatory neuronal loss in the substantia nigra induced by systemic lipopolysaccharide is prevented by knockout of the P2Y6 receptor in mice.
Journal: Journal of neuroinflammation
Published: June 14, 2021
The microglial P2Y6 receptor mediates neuronal loss and memory deficits in neurodegeneration.
Journal: Cell reports
Published: April 16, 2021
Wild-type sTREM2 blocks Aβ aggregation and neurotoxicity, but the Alzheimer's R47H mutant increases Aβ aggregation.
Journal: The Journal of biological chemistry
Published: February 05, 2021
Galectin-3, a novel endogenous TREM2 ligand, detrimentally regulates inflammatory response in Alzheimer's disease.
Journal: Acta neuropathologica
Published: November 23, 2018
Effective Knockdown of Gene Expression in Primary Microglia With siRNA and Magnetic Nanoparticles Without Cell Death or Inflammation.
Journal: Frontiers in cellular neuroscience
Published: May 30, 2018
TET2 Regulates the Neuroinflammatory Response in Microglia.
Journal: Cell reports
Published: October 24, 2017
Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration.
Journal: Scientific reports
Published: July 15, 2016
Anti-CD47 antibodies induce phagocytosis of live, malignant B cells by macrophages via the Fc domain, resulting in cell death by phagoptosis.
Journal: Oncotarget
Published: December 15, 2015
Activated Microglia Desialylate and Phagocytose Cells via Neuraminidase, Galectin-3, and Mer Tyrosine Kinase.
Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: December 03, 2015
Last Updated: 09/26/2026