Anne Vincent

Nice, France

Last Updated: 09/26/2026

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Found 14 publications

Mesenchymal stromal cells for improvement of cardiac function following acute myocardial infarction: a matter of timing.

Mesenchymal stromal cells for improvement of cardiac function following acute myocardial infarction: a matter of timing.

Journal: Physiological reviews
Published: August 17, 2023

Heart rate reduction after genetic ablation of L-type Cav1.3 channels induces cardioprotection against ischemia-reperfusion injury.

Heart rate reduction after genetic ablation of L-type Cav1.3 channels induces cardioprotection against ischemia-reperfusion injury.

Journal: Frontiers in cardiovascular medicine
Published: December 30, 2022

Correction: PPARβ/δ priming enhances the anti-apoptotic and therapeutic properties of mesenchymal stromal cells in myocardial ischemia-reperfusion injury.

Correction: PPARβ/δ priming enhances the anti-apoptotic and therapeutic properties of mesenchymal stromal cells in myocardial ischemia-reperfusion injury.

Journal: Stem cell research & therapy
Published: July 26, 2022

PPARβ/δ priming enhances the anti-apoptotic and therapeutic properties of mesenchymal stromal cells in myocardial ischemia-reperfusion injury.

PPARβ/δ priming enhances the anti-apoptotic and therapeutic properties of mesenchymal stromal cells in myocardial ischemia-reperfusion injury.

Journal: Stem cell research & therapy
Published: December 06, 2021

PPARβ/δ Is Required for Mesenchymal Stem Cell Cardioprotective Effects Independently of Their Anti-inflammatory Properties in Myocardial Ischemia-Reperfusion Injury.

PPARβ/δ Is Required for Mesenchymal Stem Cell Cardioprotective Effects Independently of Their Anti-inflammatory Properties in Myocardial Ischemia-Reperfusion Injury.

Journal: Frontiers in cardiovascular medicine
Published: March 15, 2021

Anti-apoptotic peptide for long term cardioprotection in a mouse model of myocardial ischemia-reperfusion injury.

Anti-apoptotic peptide for long term cardioprotection in a mouse model of myocardial ischemia-reperfusion injury.

Journal: Scientific reports
Published: June 24, 2020

Inhibition of G protein-gated K+ channels by tertiapin-Q rescues sinus node dysfunction and atrioventricular conduction in mouse models of primary bradycardia.

Inhibition of G protein-gated K+ channels by tertiapin-Q rescues sinus node dysfunction and atrioventricular conduction in mouse models of primary bradycardia.

Journal: Scientific reports
Published: January 24, 2020

A novel therapeutic peptide targeting myocardial reperfusion injury.

A novel therapeutic peptide targeting myocardial reperfusion injury.

Journal: Cardiovascular research
Published: July 03, 2018

Acute and long-term cardioprotective effects of the Traditional Chinese Medicine MLC901 against myocardial ischemia-reperfusion injury in mice.

Acute and long-term cardioprotective effects of the Traditional Chinese Medicine MLC901 against myocardial ischemia-reperfusion injury in mice.

Journal: Scientific reports
Published: August 11, 2017

Neuroanatomical distribution and function of the vasopressin V1B receptor in the rat brain deciphered using specific fluorescent ligands.

Neuroanatomical distribution and function of the vasopressin V1B receptor in the rat brain deciphered using specific fluorescent ligands.

Journal: General and comparative endocrinology
Published: May 03, 2017

Cardiac mGluR1 metabotropic receptors in cardioprotection.

Cardiac mGluR1 metabotropic receptors in cardioprotection.

Journal: Cardiovascular research
Published: August 04, 2016

Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation.

Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivation.

Journal: Nature communications
Published: May 07, 2014
Showing 1-12 of 14

Last Updated: 09/26/2026