Last Updated: 09/26/2026
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Found 13 publications
Soluble factors released from fibroblast-like synoviocytes of JIA patients with future extended course can condition persistent JIA synovial fibroblasts for increased IL-6 production.
Journal: Arthritis research & therapy
Published: June 11, 2025
Pathogenesis of juvenile idiopathic arthritis.
Journal: Current opinion in rheumatology
Published: May 06, 2025
Adalimumab Effectively Decreases Inflammation Downstream of TNFα Signaling in Synoviocytes from Extended Oligoarticular Juvenile Idiopathic Arthritis.
Journal: Rheumatology and therapy
Published: September 21, 2023
An Atypical Case of Scurvy in an Adolescent With Sacroiliitis.
Journal: Clinical pediatrics
Published: June 24, 2022
Single-cell analysis reveals heterogeneity of juvenile idiopathic arthritis fibroblast-like synoviocytes with implications for disease subtype.
Journal: Arthritis research & therapy
Published: June 07, 2022
Juvenile idiopathic arthritis fibroblast-like synoviocytes influence chondrocytes to alter BMP antagonist expression demonstrating an interaction between the two prominent cell types involved in endochondral bone formation.
Journal: Pediatric rheumatology online journal
Published: May 06, 2020
Distinct interferon signatures and cytokine patterns define additional systemic autoinflammatory diseases.
Journal: The Journal of clinical investigation
Published: April 02, 2019
Prior to extension, Transcriptomes of fibroblast-like Synoviocytes from extended and Polyarticular juvenile idiopathic arthritis are indistinguishable.
Journal: Pediatric rheumatology online journal
Published: September 21, 2017
Unilateral Periorbital Swelling in Two Previously Healthy Females.
Journal: Global pediatric health
Published: July 03, 2017
Secretion of pro-inflammatory cytokines and chemokines and loss of regulatory signals by fibroblast-like synoviocytes in juvenile idiopathic arthritis.
Journal: Proteomics. Clinical applications
Published: June 22, 2016
The role of transforming growth factor β signaling in fibroblast-like synoviocytes from patients with oligoarticular juvenile idiopathic arthritis: dysregulation of transforming growth factor β signaling, including overexpression of bone morphogenetic protein 4, may lead to a chondrocyte phenotype and may contribute to bony hypertrophy.
Journal: Arthritis & rheumatology (Hoboken, N.J.)
Published: March 11, 2013
Last Updated: 09/26/2026