Anthony Laugeray

UMR7355 CNRS, 3b, Rue De La Férollerie
Orleans, 45071, France

Last Updated: 09/26/2026

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Found 13 publications

The herbicides glyphosate and glufosinate and the cyanotoxin β-N-methylamino-l-alanine induce long-term motor disorders following postnatal exposure: the importance of prior asymptomatic maternal inflammatory sensitization.

The herbicides glyphosate and glufosinate and the cyanotoxin β-N-methylamino-l-alanine induce long-term motor disorders following postnatal exposure: the importance of prior asymptomatic maternal inflammatory sensitization.

Journal: Frontiers in neuroscience

Published: March 01, 2023

Synaptic Plasticity Dysfunctions in the Pathophysiology of 22q11 Deletion Syndrome: Is There a Role for Astrocytes?

Synaptic Plasticity Dysfunctions in the Pathophysiology of 22q11 Deletion Syndrome: Is There a Role for Astrocytes?

Journal: International journal of molecular sciences

Published: March 15, 2022

Disruption of Astrocyte-Dependent Dopamine Control in the Developing Medial Prefrontal Cortex Leads to Excessive Grooming in Mice.

Disruption of Astrocyte-Dependent Dopamine Control in the Developing Medial Prefrontal Cortex Leads to Excessive Grooming in Mice.

Journal: Biological psychiatry

Published: February 19, 2022

3Rs-based optimization of mice behavioral testing: The habituation/dishabituation olfactory test.

3Rs-based optimization of mice behavioral testing: The habituation/dishabituation olfactory test.

Journal: Journal of neuroscience methods

Published: September 09, 2019

Multiple effects of the herbicide glufosinate-ammonium and its main metabolite on neural stem cells from the subventricular zone of newborn mice.

Multiple effects of the herbicide glufosinate-ammonium and its main metabolite on neural stem cells from the subventricular zone of newborn mice.

Journal: Neurotoxicology

Published: May 17, 2018

Perinatal Exposure to the Cyanotoxin β-N-Méthylamino-L-Alanine (BMAA) Results in Long-Lasting Behavioral Changes in Offspring-Potential Involvement of DNA Damage and Oxidative Stress.

Perinatal Exposure to the Cyanotoxin β-N-Méthylamino-L-Alanine (BMAA) Results in Long-Lasting Behavioral Changes in Offspring-Potential Involvement of DNA Damage and Oxidative Stress.

Journal: Neurotoxicity research

Published: April 04, 2017

In utero and lactational exposure to low-doses of the pyrethroid insecticide cypermethrin leads to neurodevelopmental defects in male mice-An ethological and transcriptomic study.

In utero and lactational exposure to low-doses of the pyrethroid insecticide cypermethrin leads to neurodevelopmental defects in male mice-An ethological and transcriptomic study.

Journal: PloS one

Published: March 31, 2017

IL-33 receptor ST2 regulates the cognitive impairments associated with experimental cerebral malaria.

IL-33 receptor ST2 regulates the cognitive impairments associated with experimental cerebral malaria.

Journal: PLoS pathogens

Published: October 04, 2016

Chronic Treatment with the IDO1 Inhibitor 1-Methyl-D-Tryptophan Minimizes the Behavioural and Biochemical Abnormalities Induced by Unpredictable Chronic Mild Stress in Mice - Comparison with Fluoxetine.

Chronic Treatment with the IDO1 Inhibitor 1-Methyl-D-Tryptophan Minimizes the Behavioural and Biochemical Abnormalities Induced by Unpredictable Chronic Mild Stress in Mice - Comparison with Fluoxetine.

Journal: PloS one

Published: May 17, 2016

Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization.

Perinatal Exposure to Glufosinate Ammonium Herbicide Impairs Neurogenesis and Neuroblast Migration through Cytoskeleton Destabilization.

Journal: Frontiers in cellular neuroscience

Published: May 10, 2016

Pre- and postnatal exposure to low dose glufosinate ammonium induces autism-like phenotypes in mice.

Pre- and postnatal exposure to low dose glufosinate ammonium induces autism-like phenotypes in mice.

Journal: Frontiers in behavioral neuroscience

Published: September 19, 2014

Rescue of fragile X syndrome phenotypes in Fmr1 KO mice by a BKCa channel opener molecule.

Rescue of fragile X syndrome phenotypes in Fmr1 KO mice by a BKCa channel opener molecule.

Journal: Orphanet journal of rare diseases

Published: February 10, 2014

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Last Updated: 09/26/2026