Antonio G. De Diego

Madrid, MD, Spain

Last Updated: 09/26/2026

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Found 19 publications

Mitochondrial dysfunction in chromaffin cells from the R6/1 mouse model of Huntington's disease: Impact on exocytosis and calcium current regulation.

Mitochondrial dysfunction in chromaffin cells from the R6/1 mouse model of Huntington's disease: Impact on exocytosis and calcium current regulation.

Journal: Neurobiology of disease

Published: October 25, 2022

Combined drug triads for synergic neuroprotection in retinal degeneration.

Combined drug triads for synergic neuroprotection in retinal degeneration.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: February 20, 2022

Synthesis and Pharmacological Evaluation of Novel Non-nucleotide Purine Derivatives as P2X7 Antagonists for the Treatment of Neuroinflammation.

Synthesis and Pharmacological Evaluation of Novel Non-nucleotide Purine Derivatives as P2X7 Antagonists for the Treatment of Neuroinflammation.

Journal: Journal of medicinal chemistry

Published: February 09, 2021

Disruption of Exocytosis in Sympathoadrenal Chromaffin Cells from Mouse Models of Neurodegenerative Diseases.

Disruption of Exocytosis in Sympathoadrenal Chromaffin Cells from Mouse Models of Neurodegenerative Diseases.

Journal: International journal of molecular sciences

Published: February 04, 2020

The purinergic P2X7 receptor as a potential drug target to combat neuroinflammation in neurodegenerative diseases.

The purinergic P2X7 receptor as a potential drug target to combat neuroinflammation in neurodegenerative diseases.

Journal: Medicinal research reviews

Published: October 07, 2019

Otilonium and pinaverium trigger mitochondrial-mediated apoptosis in rat embryo cortical neurons in vitro.

Otilonium and pinaverium trigger mitochondrial-mediated apoptosis in rat embryo cortical neurons in vitro.

Journal: Neurotoxicology

Published: July 20, 2018

Altered excitability and exocytosis in chromaffin cells from the R6/1 mouse model of Huntington's disease is linked to over-expression of mutated huntingtin.

Altered excitability and exocytosis in chromaffin cells from the R6/1 mouse model of Huntington's disease is linked to over-expression of mutated huntingtin.

Journal: Journal of neurochemistry

Published: June 05, 2018

Hydrogen sulphide facilitates exocytosis by regulating the handling of intracellular calcium by chromaffin cells.

Hydrogen sulphide facilitates exocytosis by regulating the handling of intracellular calcium by chromaffin cells.

Journal: Pflugers Archiv : European journal of physiology

Published: October 30, 2017

Altered exocytosis in chromaffin cells from mouse models of neurodegenerative diseases.

Altered exocytosis in chromaffin cells from mouse models of neurodegenerative diseases.

Journal: Acta physiologica (Oxford, England)

Published: August 31, 2017

Calcium Channel Subtypes and Exocytosis in Chromaffin Cells at Early Life.

Calcium Channel Subtypes and Exocytosis in Chromaffin Cells at Early Life.

Journal: Current molecular pharmacology

Published: December 03, 2014

Depressed excitability and ion currents linked to slow exocytotic fusion pore in chromaffin cells of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Depressed excitability and ion currents linked to slow exocytotic fusion pore in chromaffin cells of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Journal: American journal of physiology. Cell physiology

Published: November 08, 2014

Hypoxia-elicited catecholamine release is controlled by L-type as well as N/PQ types of calcium channels in rat embryo chromaffin cells.

Hypoxia-elicited catecholamine release is controlled by L-type as well as N/PQ types of calcium channels in rat embryo chromaffin cells.

Journal: American journal of physiology. Cell physiology

Published: July 04, 2014

Showing 1-12 of 19

Last Updated: 09/26/2026