Anu Kauppinen

Journal: European journal of pharmaceutics and biopharmaceutics : official journal of Arbeitsgemeinschaft fur Pharmazeutische Verfahrenstechnik e.V

Published: January 09, 2025

Fibrotic Changes in Rhegmatogenous Retinal Detachment.

Journal: International journal of molecular sciences

Published: December 13, 2024

Metformin Alleviates Inflammation and Induces Mitophagy in Human Retinal Pigment Epithelium Cells Suffering from Mitochondrial Damage.

Journal: Cells

Published: July 16, 2024

Epithelial-mesenchymal Transition (EMT) and the Effect of Atorvastatin on it in ARPE-19 cells.

Journal: Cell biochemistry and biophysics

Published: May 07, 2024

Endosymbiotic theory of aging revisited: Age-related leakage of mitochondrial dsDNA/RNA stimulates cytosolic nucleic acid sensors which remodel the immune network and promote the aging process.

Endosymbiotic theory of aging revisited: Age-related leakage of mitochondrial dsDNA/RNA stimulates cytosolic nucleic acid sensors which remodel the immune network and promote the aging process.

The role of Wnt signalling in the pathogenesis and therapy of age-related macular degeneration (AMD).

The role of Wnt signalling in the pathogenesis and therapy of age-related macular degeneration (AMD).

Thapsigargin-induced autophagic flux impairment and inflammation are potentiated by CLN3 deficiency and alleviated by 5-aminoimidazole-4-carboxamide ribonucleoside (AICAR) in human ARPE-19 cells.

Thapsigargin-induced autophagic flux impairment and inflammation are potentiated by CLN3 deficiency and alleviated by 5-aminoimidazole-4-carboxamide ribonucleoside (AICAR) in human ARPE-19 cells.

Myeloid cell nuclear differentiation antigen (MNDA) is an interferon-inducible regulator of myelopoiesis. Potential role in the myeloid-biased aging process.

Myeloid cell nuclear differentiation antigen (MNDA) is an interferon-inducible regulator of myelopoiesis. Potential role in the myeloid-biased aging process.

Aberrant angiogenesis, inflammation, and fibrosis in pediatric vitreoretinal surgical diseases.

Aberrant angiogenesis, inflammation, and fibrosis in pediatric vitreoretinal surgical diseases.

Progressive rod bipolar cell loss, ubiquitin accumulation, and subretinal drusenoid deposits in Cln3Δex7/8 mice: New insights into juvenile neuronal ceroid lipofuscinosis pathogenesis.

Progressive rod bipolar cell loss, ubiquitin accumulation, and subretinal drusenoid deposits in Cln3Δex7/8 mice: New insights into juvenile neuronal ceroid lipofuscinosis pathogenesis.

Activation of cGAS-STING signaling in senescent cells promotes the aging process by remodeling the functions of the immune system.

Activation of cGAS-STING signaling in senescent cells promotes the aging process by remodeling the functions of the immune system.

Photoaging: UV radiation-induced cGAS-STING signaling promotes the aging process in skin by remodeling the immune network.

Photoaging: UV radiation-induced cGAS-STING signaling promotes the aging process in skin by remodeling the immune network.

Amino acid prodrug of capsaicin improves pharmacokinetic properties in the mouse brain and pancreas.

Amino acid prodrug of capsaicin improves pharmacokinetic properties in the mouse brain and pancreas.

Fibrotic Changes in Rhegmatogenous Retinal Detachment.

Fibrotic Changes in Rhegmatogenous Retinal Detachment.

Metformin Alleviates Inflammation and Induces Mitophagy in Human Retinal Pigment Epithelium Cells Suffering from Mitochondrial Damage.

Metformin Alleviates Inflammation and Induces Mitophagy in Human Retinal Pigment Epithelium Cells Suffering from Mitochondrial Damage.

Epithelial-mesenchymal Transition (EMT) and the Effect of Atorvastatin on it in ARPE-19 cells.

Epithelial-mesenchymal Transition (EMT) and the Effect of Atorvastatin on it in ARPE-19 cells.

Thapsigargin-induced autophagic flux impairment and inflammation are potentiated by CLN3 deficiency and alleviated by 5-aminoimidazole-4-carboxamide ribonucleoside (AICAR) in human ARPE-19 cells.

Thapsigargin-induced autophagic flux impairment and inflammation are potentiated by CLN3 deficiency and alleviated by 5-aminoimidazole-4-carboxamide ribonucleoside (AICAR) in human ARPE-19 cells.