Arisa Takeuchi
Tokyo, Japan
Last Updated: 09/26/2026
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Found 17 publications
Tokishakuyakusan suppresses endometriosis and may be associated with improved fertility during attempts to conceive: A mouse model study.
Journal: Journal of reproductive immunology
Published: March 21, 2026
Impact of Chronic Exposure to Endometriosis on Perinatal Outcomes: Establishment of a Mouse Model.
Journal: Biomedicines
Published: August 28, 2022
Establishment of a novel mouse model of adenomyosis suitable for longitudinal and quantitative analysis and perinatal outcome studies.
Journal: Scientific reports
Published: February 16, 2022
The roles of polymorphonuclear myeloid-derived suppressor cells in endometriosis.
Journal: Journal of reproductive immunology
Published: June 13, 2021
Use of selective PGE2 receptor antagonists on human endometriotic stromal cells and peritoneal macrophages.
Journal: Molecular human reproduction
Published: July 06, 2020
IFITM1 is a Novel, Highly Sensitive Marker for Endometriotic Stromal Cells in Ovarian and Extragenital Endometriosis.
Journal: Reproductive sciences (Thousand Oaks, Calif.)
Published: May 22, 2020
PAX8: A Highly Sensitive Marker for the Glands in Extragenital Endometriosis.
Journal: Reproductive sciences (Thousand Oaks, Calif.)
Published: May 21, 2020
Activation of endoplasmic reticulum stress mediates oxidative stress-induced apoptosis of granulosa cells in ovaries affected by endometrioma.
Journal: Molecular human reproduction
Published: August 29, 2019
The effects of tokishakuyakusan, a traditional Japanese medicine (kampo), ferulic acid and paeoniflorin, on human endometriotic stromal cells and peritoneal macrophages.
Journal: Journal of reproductive immunology
Published: March 06, 2019
IFITM1 is a Novel, Highly Sensitive Marker for Endometriotic Stromal Cells in Ovarian and Extragenital Endometriosis.
Journal: Reproductive sciences (Thousand Oaks, Calif.)
Published: February 23, 2019
PAX8: A Highly Sensitive Marker for the Glands in Extragenital Endometriosis.
Journal: Reproductive sciences (Thousand Oaks, Calif.)
Published: February 16, 2019
Endometriosis Triggers Excessive Activation of Primordial Follicles via PI3K-PTEN-Akt-Foxo3 Pathway.
Endometriosis Triggers Excessive Activation of Primordial Follicles via PI3K-PTEN-Akt-Foxo3 Pathway.
Journal: The Journal of clinical endocrinology and metabolism
Published: February 03, 2019
Last Updated: 09/26/2026