Armelle Corpet
Paris, 75231, France
Last Updated: 09/26/2026
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Found 19 publications
Lethargy in progress! The HUSH complex controls HSV-1 latency
Journal: Medecine sciences : M/S
Published: November 19, 2025
Defective plasticity in dermatomyositis patients muscle stem cells is associated with sustained intrinsic inflammatory signaling and disruption of the histone H3.3 chromatin loading pathway.
Journal: NAR molecular medicine
Published: September 03, 2025
The HUSH epigenetic repressor complex silences PML nuclear body-associated HSV-1 quiescent genomes.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: November 26, 2024
The TUDOR domain of SMN is an H3K79me1 histone mark reader.
Journal: Life science alliance
Published: October 05, 2022
Interplay between PML NBs and HIRA for H3.3 dynamics following type I interferon stimulus.
Journal: eLife
Published: May 10, 2022
SMA-linked SMN mutants prevent phase separation properties and SMN interactions with FMRP family members.
Journal: Life science alliance
Published: February 28, 2022
PML nuclear bodies and chromatin dynamics: catch me if you can!
Journal: Nucleic acids research
Published: June 11, 2020
Latent/Quiescent Herpes Simplex Virus 1 Genome Detection by Fluorescence In Situ Hybridization (FISH).
Journal: Methods in molecular biology (Clifton, N.J.)
Published: October 17, 2019
Promyelocytic leukemia (PML) nuclear bodies (NBs) induce latent/quiescent HSV-1 genomes chromatinization through a PML NB/Histone H3.3/H3.3 Chaperone Axis.
Journal: PLoS pathogens
Published: August 09, 2018
Last Updated: 09/26/2026