Artur Zajkowicz

Gliwice Branch
Gliwice, SL, Poland

Last Updated: 09/26/2026

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Found 11 publications

Pathogenic mobile element insertion in the MEN1 gene mimicking a deletion in MLPA: characterisation by long-read sequencing.

Pathogenic mobile element insertion in the MEN1 gene mimicking a deletion in MLPA: characterisation by long-read sequencing.

Journal: Journal of medical genetics

Published: September 26, 2025

Are the Common Genetic 3'UTR Variants in ADME Genes Playing a Role in Tolerance of Breast Cancer Chemotherapy?

Are the Common Genetic 3'UTR Variants in ADME Genes Playing a Role in Tolerance of Breast Cancer Chemotherapy?

Journal: International journal of molecular sciences

Published: October 16, 2024

Transcriptome Analysis of Cells Exposed to Actinomycin D and Nutlin-3a Reveals New Candidate p53-Target Genes and Indicates That CHIR-98014 Is an Important Inhibitor of p53 Activity.

Transcriptome Analysis of Cells Exposed to Actinomycin D and Nutlin-3a Reveals New Candidate p53-Target Genes and Indicates That CHIR-98014 Is an Important Inhibitor of p53 Activity.

Journal: International journal of molecular sciences

Published: August 30, 2021

BRCA1/BRCA2 variants of uncertain significance in clinical practice: A case report.

BRCA1/BRCA2 variants of uncertain significance in clinical practice: A case report.

Journal: Molecular and clinical oncology

Published: July 13, 2020

Synergistic activation of p53 by actinomycin D and nutlin-3a is associated with the upregulation of crucial regulators and effectors of innate immunity.

Synergistic activation of p53 by actinomycin D and nutlin-3a is associated with the upregulation of crucial regulators and effectors of innate immunity.

Journal: Cellular signalling

Published: September 27, 2019

PIM2 survival kinase is upregulated in a p53-dependent manner in cells treated with camptothecin or co-treated with actinomycin D and nutlin-3a.

PIM2 survival kinase is upregulated in a p53-dependent manner in cells treated with camptothecin or co-treated with actinomycin D and nutlin-3a.

Journal: Archives of biochemistry and biophysics

Published: April 26, 2018

The Alzheimer's disease-associated TREM2 gene is regulated by p53 tumor suppressor protein.

The Alzheimer's disease-associated TREM2 gene is regulated by p53 tumor suppressor protein.

Journal: Neuroscience letters

Published: February 24, 2018

Actinomycin D and nutlin-3a synergistically promote phosphorylation of p53 on serine 46 in cancer cell lines of different origin.

Actinomycin D and nutlin-3a synergistically promote phosphorylation of p53 on serine 46 in cancer cell lines of different origin.

Journal: Cellular signalling

Published: February 15, 2015

Truncating mutations of PPM1D are found in blood DNA samples of lung cancer patients.

Truncating mutations of PPM1D are found in blood DNA samples of lung cancer patients.

Journal: British journal of cancer

Published: October 20, 2014

Rapamycin prevents strong phosphorylation of p53 on serine 46 and attenuates activation of the p53 pathway in A549 lung cancer cells exposed to actinomycin D.

Rapamycin prevents strong phosphorylation of p53 on serine 46 and attenuates activation of the p53 pathway in A549 lung cancer cells exposed to actinomycin D.

Journal: Mechanisms of ageing and development

Published: October 16, 2013

Nutlin-3a, an MDM2 antagonist and p53 activator, helps to preserve the replicative potential of cancer cells treated with a genotoxic dose of resveratrol.

Nutlin-3a, an MDM2 antagonist and p53 activator, helps to preserve the replicative potential of cancer cells treated with a genotoxic dose of resveratrol.

Journal: Molecular biology reports

Published: July 09, 2012

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Last Updated: 09/26/2026