Arun Lingampally
Giessen, HE, Germany
Last Updated: 09/26/2026
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Found 18 publications
Revisiting pulmonary fibrosis: inflammatory dynamics of the lipofibroblast-to-inflammatory lipofibroblast-to-activated myofibroblast reversible switch.
Journal: Frontiers in immunology
Published: April 10, 2025
Unraveling Alveolar Fibroblast and Activated Myofibroblast Heterogeneity and Differentiation Trajectories During Lung Fibrosis Development and Resolution in Young and Old Mice.
Journal: Aging cell
Published: October 21, 2024
GLI1+ Cells Contribute to Vascular Remodeling in Pulmonary Hypertension.
Journal: Circulation research
Published: April 19, 2024
Aging affects reprogramming of pulmonary capillary endothelial cells after lung injury in male mice.
Aging affects reprogramming of pulmonary capillary endothelial cells after lung injury in male mice.
Journal: Nature communications
Published: March 11, 2024
Understanding myofibroblast origin in the fibrotic lung.
Journal: Chinese medical journal pulmonary and critical care medicine
Published: February 14, 2024
Highlighting fibroblast plasticity in lung fibrosis: the WI-38 cell line as a model for investigating the myofibroblast and lipofibroblast switch.
Journal: Theranostics
Published: December 23, 2023
Myofibroblasts emerge during alveolar regeneration following influenza-virus-induced lung injury.
Journal: Cell reports
Published: December 13, 2023
Evidence for a lipofibroblast-to-Cthrc1 + myofibroblast reversible switch during the development and resolution of lung fibrosis in young mice.
Journal: The European respiratory journal
Published: March 21, 2023
FGFR2b signalling restricts lineage-flexible alveolar progenitors during mouse lung development and converges in mature alveolar type 2 cells.
Journal: Cellular and molecular life sciences : CMLS
Published: October 11, 2022
Insights into the Black Box of Intra-Amniotic Infection and Its Impact on the Premature Lung: From Clinical and Preclinical Perspectives.
Journal: International journal of molecular sciences
Published: August 08, 2022
GLI1+ cells are a source of repair-supportive mesenchymal cells (RSMCs) during airway epithelial regeneration.
Journal: Cellular and molecular life sciences : CMLS
Published: June 09, 2022
Last Updated: 09/26/2026