Aurelie Mercier - Publications

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities.

Journal: Scientific reports

Published: March 20, 2020

A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy.

Journal: Scientific reports

Published: May 11, 2018

Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.

Journal: The Canadian journal of cardiology

Published: January 26, 2016

Unmasked Brugada pattern by ajmaline challenge in patients with myotonic dystrophy type 1.

Journal: Annals of noninvasive electrocardiology : the official journal of the International Society for Holter and Noninvasive Electrocardiology, Inc

Published: June 20, 2014

Nav1.5 channels can reach the plasma membrane through distinct N-glycosylation states.

Journal: Biochimica et biophysica acta

Published: June 16, 2014

Myotonic dystrophy type 1 mimics and exacerbates Brugada phenotype induced by Nav1.5 sodium channel loss-of-function mutation.

Journal: Heart rhythm

Published: September 29, 2013

A distinct de novo expression of Nav1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts.

Journal: The Journal of physiology

Published: July 18, 2012

The β1-subunit of Na(v)1.5 cardiac sodium channel is required for a dominant negative effect through α-α interaction.

Journal: PloS one

Published: July 12, 2012