Aya Ishida-Ito
Tokyo, 1608582, Japan
Last Updated: 09/26/2026
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Found 12 publications
Synaptic disturbance in neurodevelopmental disorders: Perspectives from fragile X and Rett syndromes.
Journal: Brain & development
Published: January 23, 2025
Loss of MeCP2 Function Across Several Neuronal Populations Impairs Breathing Response to Acute Hypoxia.
Journal: Frontiers in neurology
Published: August 10, 2020
MeCP2 Levels Regulate the 3D Structure of Heterochromatic Foci in Mouse Neurons.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: June 02, 2019
Improvement of cerebellar ataxic gait by injecting Cbln1 into the cerebellum of cbln1-null mice.
Journal: Scientific reports
Published: November 15, 2017
Genome-wide distribution of linker histone H1.0 is independent of MeCP2.
Journal: Nature neuroscience
Published: February 13, 2017
Restoration of Mecp2 expression in GABAergic neurons is sufficient to rescue multiple disease features in a mouse model of Rett syndrome.
Journal: eLife
Published: January 04, 2016
Loss and Gain of MeCP2 Cause Similar Hippocampal Circuit Dysfunction that Is Rescued by Deep Brain Stimulation in a Rett Syndrome Mouse Model.
Journal: Neuron
Published: March 08, 2015
The role of Cbln1 on Purkinje cell synapse formation.
Journal: Neuroscience research
Published: January 07, 2014
Reevaluation of the role of parallel fiber synapses in delay eyeblink conditioning in mice using Cbln1 as a tool.
Journal: Frontiers in neural circuits
Published: August 10, 2013
Cbln1 downregulates the formation and function of inhibitory synapses in mouse cerebellar Purkinje cells.
Journal: The European journal of neuroscience
Published: March 21, 2013
Presynaptically released Cbln1 induces dynamic axonal structural changes by interacting with GluD2 during cerebellar synapse formation.
Journal: Neuron
Published: July 30, 2012
Showing 1-12 of 12
Last Updated: 09/26/2026