Beate Hagl

Dr. Von Haunersches Kinderspital
Munich, BY 80337, Germany

Last Updated: 09/26/2026

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Found 21 publications

Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer Formation.

Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer Formation.

Journal: European journal of immunology

Published: June 06, 2024

Staphylococcus aureus Serine protease-like protein A (SplA) induces IL-8 by keratinocytes and synergizes with IL-17A.

Staphylococcus aureus Serine protease-like protein A (SplA) induces IL-8 by keratinocytes and synergizes with IL-17A.

Journal: Cytokine

Published: May 13, 2023

Molecular Assessment of Staphylococcus Aureus Strains in STAT3 Hyper-IgE Syndrome Patients.

Molecular Assessment of Staphylococcus Aureus Strains in STAT3 Hyper-IgE Syndrome Patients.

Journal: Journal of clinical immunology

Published: January 07, 2022

Rescue of STAT3 Function in Hyper-IgE Syndrome Using Adenine Base Editing.

Rescue of STAT3 Function in Hyper-IgE Syndrome Using Adenine Base Editing.

Journal: The CRISPR journal

Published: April 20, 2021

Class Switch Recombination Defects: impact on B cell maturation and antibody responses.

Class Switch Recombination Defects: impact on B cell maturation and antibody responses.

Journal: Clinical immunology (Orlando, Fla.)

Published: June 16, 2020

Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential.

Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential.

Journal: Orphanet journal of rare diseases

Published: February 22, 2020

Downregulation of SFRP1 is a protumorigenic event in hepatoblastoma and correlates with beta-catenin mutations.

Downregulation of SFRP1 is a protumorigenic event in hepatoblastoma and correlates with beta-catenin mutations.

Journal: Journal of cancer research and clinical oncology

Published: October 25, 2019

Impaired memory B-cell development and antibody maturation with a skewing toward IgE in patients with STAT3 hyper-IgE syndrome.

Impaired memory B-cell development and antibody maturation with a skewing toward IgE in patients with STAT3 hyper-IgE syndrome.

Journal: Allergy

Published: September 05, 2018

Somatic alterations compromised molecular diagnosis of DOCK8 hyper-IgE syndrome caused by a novel intronic splice site mutation.

Somatic alterations compromised molecular diagnosis of DOCK8 hyper-IgE syndrome caused by a novel intronic splice site mutation.

Journal: Scientific reports

Published: June 25, 2018

Lung disease in STAT3 hyper-IgE syndrome requires intense therapy.

Lung disease in STAT3 hyper-IgE syndrome requires intense therapy.

Journal: Allergy

Published: May 03, 2018

Atopic eczema in childhood or primary immunodeficiency - what needs to be considered?

Atopic eczema in childhood or primary immunodeficiency - what needs to be considered?

Journal: MMW Fortschritte der Medizin

Published: November 22, 2017

STAT1 Gain-of-Function and Dominant Negative STAT3 Mutations Impair IL-17 and IL-22 Immunity Associated with CMC.

STAT1 Gain-of-Function and Dominant Negative STAT3 Mutations Impair IL-17 and IL-22 Immunity Associated with CMC.

Journal: The Journal of investigative dermatology

Published: February 21, 2017

Showing 1-12 of 21

Last Updated: 09/26/2026