Beatriz Guzman-Flores
Auckland, AUK, New Zealand
Last Updated: 09/26/2026
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Found 9 publications
The β-amyloid-induced increased tonic conductance, impaired long-term potentiation and cognitive deficits characteristic of Alzheimer's disease are reversed by an α5 inverse agonist of the GABA type A receptor.
Journal: Neuropharmacology
Published: September 04, 2025
A novel allosteric GCase modulator prevents Tau accumulation in GBA1WT and GBA1L444P/L444P cellular models.
Journal: Scientific reports
Published: October 26, 2024
Amyloid-beta1-42 induced glutamatergic receptor and transporter expression changes in the mouse hippocampus.
Journal: Journal of neurochemistry
Published: May 01, 2020
The Interplay Between Beta-Amyloid 1-42 (Aβ1-42)-Induced Hippocampal Inflammatory Response, p-tau, Vascular Pathology, and Their Synergistic Contributions to Neuronal Death and Behavioral Deficits.
Journal: Frontiers in molecular neuroscience
Published: April 15, 2020
Amyloid-Beta1-42 -Induced Increase in GABAergic Tonic Conductance in Mouse Hippocampal CA1 Pyramidal Cells.
Journal: Molecules (Basel, Switzerland)
Published: November 28, 2019
Gamma-aminobutyric acid A receptors in Alzheimer's disease: highly localized remodeling of a complex and diverse signaling pathway.
Journal: Neural regeneration research
Published: August 15, 2018
The GABAergic system as a therapeutic target for Alzheimer's disease.
Journal: Journal of neurochemistry
Published: October 18, 2017
GABAA receptor subunit expression changes in the human Alzheimer's disease hippocampus, subiculum, entorhinal cortex and superior temporal gyrus.
Journal: Journal of neurochemistry
Published: October 09, 2017
Towards a Better Understanding of GABAergic Remodeling in Alzheimer's Disease.
Journal: International journal of molecular sciences
Published: July 31, 2017
Showing 1-9 of 9
Last Updated: 09/26/2026