Benedetta Terragni
Milan, 20133, Italy
Last Updated: 09/26/2026
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Found 12 publications
Histone Deacetylase Inhibitors Ameliorate Morphological Defects and Hypoexcitability of iPSC-Neurons from Rubinstein-Taybi Patients.
Journal: International journal of molecular sciences
Published: April 23, 2021
SULT4A1 Modulates Synaptic Development and Function by Promoting the Formation of PSD-95/NMDAR Complex.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: September 11, 2019
Hyperexcitability in Cultured Cortical Neuron Networks from the G93A-SOD1 Amyotrophic Lateral Sclerosis Model Mouse and its Molecular Correlates.
Journal: Neuroscience
Published: February 19, 2019
DNA damage and transcriptional regulation in iPSC-derived neurons from Ataxia Telangiectasia patients.
Journal: Scientific reports
Published: September 06, 2018
iPSC-derived neurons of CREBBP- and EP300-mutated Rubinstein-Taybi syndrome patients show morphological alterations and hypoexcitability.
Journal: Stem cell research
Published: December 11, 2017
Rare coding variants in genes encoding GABAA receptors in genetic generalised epilepsies: an exome-based case-control study.
Journal: The Lancet. Neurology
Published: August 02, 2017
The impact of genetic and experimental studies on classification and therapy of the epilepsies.
Journal: Neuroscience letters
Published: February 28, 2017
Post-translational dysfunctions in channelopathies of the nervous system.
Journal: Neuropharmacology
Published: February 14, 2017
The X-Linked Intellectual Disability Protein IL1RAPL1 Regulates Dendrite Complexity.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: December 09, 2016
Ranolazine vs phenytoin: greater effect of ranolazine on the transient Na(+) current than on the persistent Na(+) current in central neurons.
Journal: Neuropharmacology
Published: March 11, 2016
A caveolin-binding domain in the HCN4 channels mediates functional interaction with caveolin proteins.
Journal: Journal of molecular and cellular cardiology
Published: January 13, 2012
Recessive loss-of-function mutation in the pacemaker HCN2 channel causing increased neuronal excitability in a patient with idiopathic generalized epilepsy.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: December 02, 2011
Showing 1-12 of 12
Last Updated: 09/26/2026