Benjamin H. Gern

Infectious Disease
4800 Sand Point Way NE
Seattle, WA 98105

Last Updated: 09/26/2026

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Found 21 publications

Modulation of Ferroptosis During Early Mycobacterium tuberculosis Infection Contributes to Beijing Lineage Strain SA161 Virulence.

Modulation of Ferroptosis During Early Mycobacterium tuberculosis Infection Contributes to Beijing Lineage Strain SA161 Virulence.

Journal: bioRxiv : the preprint server for biology
Published: June 12, 2026

Single-cell profiling reveals that dynamic lung immune responses distinguish protection from susceptibility to tuberculosis.

Single-cell profiling reveals that dynamic lung immune responses distinguish protection from susceptibility to tuberculosis.

Journal: PLoS pathogens
Published: October 14, 2025

Monocytic niches escape T cell surveillance and promote Mycobacterium tuberculosis persistence in lymph nodes.

Monocytic niches escape T cell surveillance and promote Mycobacterium tuberculosis persistence in lymph nodes.

Journal: Immunity
Published: October 06, 2025

Expression of CD103 facilitates localization and activation of CD4+ T cells within Mycobacterium tuberculosis lung-lesions.

Expression of CD103 facilitates localization and activation of CD4+ T cells within Mycobacterium tuberculosis lung-lesions.

Journal: Mucosal immunology
Published: August 18, 2025

Early and opposing neutrophil and CD4 T cell responses shape pulmonary tuberculosis pathology.

Early and opposing neutrophil and CD4 T cell responses shape pulmonary tuberculosis pathology.

Journal: The Journal of experimental medicine
Published: January 23, 2025

APOE Protects Against Severe Infection with Mycobacterium tuberculosis by Restraining Production of Neutrophil Extracellular Traps.

APOE Protects Against Severe Infection with Mycobacterium tuberculosis by Restraining Production of Neutrophil Extracellular Traps.

Journal: bioRxiv : the preprint server for biology
Published: November 28, 2024

APOE protects against severe infection with Mycobacterium tuberculosis by restraining production of neutrophil extracellular traps.

APOE protects against severe infection with Mycobacterium tuberculosis by restraining production of neutrophil extracellular traps.

Journal: PLoS pathogens
Published: October 10, 2024

Host and pathogen genetic diversity shape vaccine-mediated protection to Mycobacterium tuberculosis.

Host and pathogen genetic diversity shape vaccine-mediated protection to Mycobacterium tuberculosis.

Journal: Frontiers in immunology
Published: May 04, 2024

CD4-mediated immunity shapes neutrophil-driven tuberculous pathology.

CD4-mediated immunity shapes neutrophil-driven tuberculous pathology.

Journal: bioRxiv : the preprint server for biology
Published: April 25, 2024

Re-appraising the role of T-cell derived interferon gamma in restriction of Mycobacterium tuberculosis in the murine lung: T-cell derived IFNγ is required to restrict pulmonary Mtb.

Re-appraising the role of T-cell derived interferon gamma in restriction of Mycobacterium tuberculosis in the murine lung: T-cell derived IFNγ is required to restrict pulmonary Mtb.

Journal: bioRxiv : the preprint server for biology
Published: April 15, 2024

Reappraising the Role of T Cell-Derived IFN-γ in Restriction of Mycobacterium tuberculosis in the Murine Lung.

Reappraising the Role of T Cell-Derived IFN-γ in Restriction of Mycobacterium tuberculosis in the Murine Lung.

Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: March 14, 2024

TOLLIP inhibits lipid accumulation and the integrated stress response in alveolar macrophages to control Mycobacterium tuberculosis infection.

TOLLIP inhibits lipid accumulation and the integrated stress response in alveolar macrophages to control Mycobacterium tuberculosis infection.

Journal: Nature microbiology
Published: February 15, 2022
Showing 1-12 of 21

Last Updated: 09/26/2026