Benoit Denhez
Sherbrooke, QC, Canada
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 12 publications
Erratum to "Deletion of protein tyrosine phosphatase SHP-1 restores SUMOylation of podocin and reverses the progression of diabetic kidney disease." Kidney Int. 2023;104:787-802.
Journal: Kidney international
Published: November 19, 2023
Deletion of protein tyrosine phosphatase SHP-1 restores SUMOylation of podocin and reverses the progression of diabetic kidney disease.
Journal: Kidney international
Published: December 16, 2022
Reduction of DUSP4 contributes to podocytes oxidative stress, insulin resistance and diabetic nephropathy.
Journal: Biochemical and biophysical research communications
Published: July 13, 2022
Interlaboratory bias of albuminuria and proteinuria in hypertensive pregnancy.
Journal: Clinical biochemistry
Published: June 29, 2020
Saturated fatty acids induce insulin resistance in podocytes through inhibition of IRS1 via activation of both IKKβ and mTORC1.
Journal: Scientific reports
Published: March 26, 2020
Regulation of Nephrin Phosphorylation in Diabetes and Chronic Kidney Injury.
Journal: Advances in experimental medicine and biology
Published: June 23, 2017
Persistent Insulin Resistance in Podocytes Caused by Epigenetic Changes of SHP-1 in Diabetes.
Journal: Diabetes
Published: February 23, 2016
Increased SHP-1 protein expression by high glucose levels reduces nephrin phosphorylation in podocytes.
Journal: The Journal of biological chemistry
Published: November 19, 2014
PKCδ impaired vessel formation and angiogenic factor expression in diabetic ischemic limbs.
Journal: Diabetes
Published: April 06, 2013
Expression of SHP-1 induced by hyperglycemia prevents insulin actions in podocytes.
Journal: American journal of physiology. Endocrinology and metabolism
Published: March 28, 2013
Showing 1-12 of 12
Last Updated: 09/26/2026