Bente Vilsen

Arhus, 8000, Denmark

Last Updated: 09/26/2026

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Found 33 publications

A Highly Conserved Glycine in a Hotspot for Neurological Disease Mutations in Na+,K+-ATPase Is Critical to Na+ and K+ Occlusion.

A Highly Conserved Glycine in a Hotspot for Neurological Disease Mutations in Na+,K+-ATPase Is Critical to Na+ and K+ Occlusion.

Journal: Biomolecules

Published: March 11, 2026

On the mechanism of hypomagnesemia with treatment-resistant seizures caused by variants of the Na+,K+-ATPase α1 subunit (ATP1A1).

On the mechanism of hypomagnesemia with treatment-resistant seizures caused by variants of the Na+,K+-ATPase α1 subunit (ATP1A1).

Journal: The Journal of general physiology

Published: December 22, 2025

How palytoxin transforms the Na+,K+ pump into a cation channel.

How palytoxin transforms the Na+,K+ pump into a cation channel.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: September 16, 2025

Na+,K+-ATPase with Disrupted Na+ Binding Sites I and III Binds Na+ with Increased Affinity at Site II and Undergoes Na+-Activated Phosphorylation with ATP.

Na+,K+-ATPase with Disrupted Na+ Binding Sites I and III Binds Na+ with Increased Affinity at Site II and Undergoes Na+-Activated Phosphorylation with ATP.

Journal: Biomolecules

Published: December 12, 2023

On the track of the lipid transport pathway of the phospholipid flippase ATP8A2 - Mutation analysis of residues of the transmembrane segments M1, M2, M3 and M4.

On the track of the lipid transport pathway of the phospholipid flippase ATP8A2 - Mutation analysis of residues of the transmembrane segments M1, M2, M3 and M4.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: July 03, 2023

Crystal structures of Na+ ,K+ -ATPase reveal the mechanism that converts the K+ -bound form to Na+ -bound form and opens and closes the cytoplasmic gate.

Crystal structures of Na+ ,K+ -ATPase reveal the mechanism that converts the K+ -bound form to Na+ -bound form and opens and closes the cytoplasmic gate.

Journal: FEBS letters

Published: March 14, 2023

Temperature instability of a mutation at a multidomain junction in Na,K-ATPase isoform ATP1A3 (p.Arg756His) produces a fever-induced neurological syndrome.

Temperature instability of a mutation at a multidomain junction in Na,K-ATPase isoform ATP1A3 (p.Arg756His) produces a fever-induced neurological syndrome.

Journal: The Journal of biological chemistry

Published: July 01, 2022

Cryo-electron microscopy of Na+ ,K+ -ATPase reveals how the extracellular gate locks in the E2·2K+ state.

Cryo-electron microscopy of Na+ ,K+ -ATPase reveals how the extracellular gate locks in the E2·2K+ state.

Journal: FEBS letters

Published: May 16, 2022

Cryoelectron microscopy of Na+,K+-ATPase in the two E2P states with and without cardiotonic steroids.

Cryoelectron microscopy of Na+,K+-ATPase in the two E2P states with and without cardiotonic steroids.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: April 05, 2022

Role of a conserved ion-binding site tyrosine in ion selectivity of the Na+/K+ pump.

Role of a conserved ion-binding site tyrosine in ion selectivity of the Na+/K+ pump.

Journal: The Journal of general physiology

Published: October 19, 2021

Binding of cardiotonic steroids to Na+,K+-ATPase in the E2P state.

Binding of cardiotonic steroids to Na+,K+-ATPase in the E2P state.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: December 15, 2020

ATP1A2- and ATP1A3-associated early profound epileptic encephalopathy and polymicrogyria.

ATP1A2- and ATP1A3-associated early profound epileptic encephalopathy and polymicrogyria.

Journal: Brain : a journal of neurology

Published: August 14, 2020

Showing 1-12 of 33

Last Updated: 09/26/2026