Beom S. Kang

Chuncheon, Republic of Korea

Last Updated: 09/26/2026

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Found 23 publications

Imipramine, an Acid Sphingomyelinase Inhibitor, Promotes Newborn Neuron Survival in the Hippocampus After Seizure.

Imipramine, an Acid Sphingomyelinase Inhibitor, Promotes Newborn Neuron Survival in the Hippocampus After Seizure.

Journal: Cells

Published: January 07, 2025

Effects of astrocytic PKM2 gene deletion on neuronal death following traumatic brain injury.

Effects of astrocytic PKM2 gene deletion on neuronal death following traumatic brain injury.

Journal: Cell death discovery

Published: August 23, 2024

L-theanine ameliorates traumatic-brain-injury-induced hippocampal neuronal death in rats.

L-theanine ameliorates traumatic-brain-injury-induced hippocampal neuronal death in rats.

Journal: Phytomedicine : international journal of phytotherapy and phytopharmacology

Published: November 17, 2023

A phosphodiesterase 4 (PDE4) inhibitor, amlexanox, reduces neuroinflammation and neuronal death after pilocarpine-induced seizure.

A phosphodiesterase 4 (PDE4) inhibitor, amlexanox, reduces neuroinflammation and neuronal death after pilocarpine-induced seizure.

Journal: Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics

Published: October 31, 2023

Pathophysiological Roles of Transient Receptor Potential (Trp) Channels and Zinc Toxicity in Brain Disease.

Pathophysiological Roles of Transient Receptor Potential (Trp) Channels and Zinc Toxicity in Brain Disease.

Journal: International journal of molecular sciences

Published: March 03, 2023

Engineered Mesenchymal Stem Cells Over-Expressing BDNF Protect the Brain from Traumatic Brain Injury-Induced Neuronal Death, Neurological Deficits, and Cognitive Impairments.

Engineered Mesenchymal Stem Cells Over-Expressing BDNF Protect the Brain from Traumatic Brain Injury-Induced Neuronal Death, Neurological Deficits, and Cognitive Impairments.

Journal: Pharmaceuticals (Basel, Switzerland)

Published: February 06, 2023

GAP-43 closely interacts with BDNF in hippocampal neurons and is associated with Alzheimer's disease progression.

GAP-43 closely interacts with BDNF in hippocampal neurons and is associated with Alzheimer's disease progression.

Journal: Frontiers in molecular neuroscience

Published: January 24, 2023

The Protective Role of Glutathione on Zinc-Induced Neuron Death after Brain Injuries.

The Protective Role of Glutathione on Zinc-Induced Neuron Death after Brain Injuries.

Journal: International journal of molecular sciences

Published: December 13, 2022

Combined Treatment of Dichloroacetic Acid and Pyruvate Increased Neuronal Survival after Seizure.

Combined Treatment of Dichloroacetic Acid and Pyruvate Increased Neuronal Survival after Seizure.

Journal: Nutrients

Published: October 31, 2022

Acid Sphingomyelinase Inhibitor, Imipramine, Reduces Hippocampal Neuronal Death after Traumatic Brain Injury.

Acid Sphingomyelinase Inhibitor, Imipramine, Reduces Hippocampal Neuronal Death after Traumatic Brain Injury.

Journal: International journal of molecular sciences

Published: October 07, 2022

The Inhibition of Zinc Excitotoxicity and AMPK Phosphorylation by a Novel Zinc Chelator, 2G11, Ameliorates Neuronal Death Induced by Global Cerebral Ischemia.

The Inhibition of Zinc Excitotoxicity and AMPK Phosphorylation by a Novel Zinc Chelator, 2G11, Ameliorates Neuronal Death Induced by Global Cerebral Ischemia.

Journal: Antioxidants (Basel, Switzerland)

Published: September 30, 2022

Administration of an Acidic Sphingomyelinase (ASMase) Inhibitor, Imipramine, Reduces Hypoglycemia-Induced Hippocampal Neuronal Death.

Administration of an Acidic Sphingomyelinase (ASMase) Inhibitor, Imipramine, Reduces Hypoglycemia-Induced Hippocampal Neuronal Death.

Journal: Cells

Published: November 30, 2021

Showing 1-12 of 23

Last Updated: 09/26/2026