Bingxiang Zhao
Dali, China
Last Updated: 09/26/2026
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Found 8 publications
Cyclosporine A ameliorates ulcerative colitis by inhibiting cellular senescence, modulating the JAK2-STAT3/NF-κB signaling pathway, and regulating the gut microbiota-metabolite axis.
Journal: International immunopharmacology
Published: September 04, 2025
Tucidinostat ameliorates DSS-induced ulcerative colitis by inhibiting cellular senescence, modulating the p53 signaling pathway and cell cycle, and restoring the gut microbiota-metabolite Axis.
Journal: International immunopharmacology
Published: August 19, 2025
Network pharmacology and experimental validation reveal Thiamphenicol ameliorates dextran sulfate sodium-induced colitis in mice via inhibition of intestinal senescence and modulation of the NF-κB/AMPK signaling pathway.
Journal: Toxicology and applied pharmacology
Published: June 18, 2025
Miconazole alleviates colitis by suppressing colonic senescence, NF-κB Signaling and gut microbiota modulation.
Journal: Toxicology and applied pharmacology
Published: June 16, 2025
Sparfloxacin ameliorates DSS-induced ulcerative colitis by suppressing cellular senescence, JAK/NF-κB signaling pathway and modulation of the gut microbiota-metabolite axis.
Journal: Biochemical pharmacology
Published: May 12, 2025
Experimental and network pharmacology certify itraconazole mitigates fluorouracil-induced intestinal damage by inhibiting mTOR-mediated intestinal senescence.
Journal: Toxicology and applied pharmacology
Published: March 20, 2025
Vorinostat attenuates UVB-induced skin senescence by modulating NF-κB and mTOR signaling pathways.
Journal: Scientific reports
Published: August 12, 2024
Ultraviolet Light Causes Skin Cell Senescence: From Mechanism to Prevention Principle.
Journal: Advanced biology
Published: February 18, 2024
Showing 1-8 of 8
Last Updated: 09/26/2026