Bo Tang

Qingpu, China

Last Updated: 09/26/2026

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Found 12 publications

Refractory idiopathic membranous nephropathy treated with rituximab combined with double filtration plasmapheresis.

Refractory idiopathic membranous nephropathy treated with rituximab combined with double filtration plasmapheresis.

Journal: Minerva surgery
Published: February 06, 2024

Exosomal hsa_circ_0125310 promotes cell proliferation and fibrosis in diabetic nephropathy via sponging miR-422a and targeting the IGF1R/p38 axis.

Exosomal hsa_circ_0125310 promotes cell proliferation and fibrosis in diabetic nephropathy via sponging miR-422a and targeting the IGF1R/p38 axis.

Journal: Journal of cellular and molecular medicine
Published: November 14, 2020

hsa-miR-199b-3p Prevents the Epithelial-Mesenchymal Transition and Dysfunction of the Renal Tubule by Regulating E-cadherin through Targeting KDM6A in Diabetic Nephropathy.

hsa-miR-199b-3p Prevents the Epithelial-Mesenchymal Transition and Dysfunction of the Renal Tubule by Regulating E-cadherin through Targeting KDM6A in Diabetic Nephropathy.

Journal: Oxidative medicine and cellular longevity
Published: September 20, 2020

Exosomal circ_DLGAP4 promotes diabetic kidney disease progression by sponging miR-143 and targeting ERBB3/NF-κB/MMP-2 axis.

Exosomal circ_DLGAP4 promotes diabetic kidney disease progression by sponging miR-143 and targeting ERBB3/NF-κB/MMP-2 axis.

Journal: Cell death & disease
Published: May 09, 2020

Loss of lncRNA MIAT ameliorates proliferation and fibrosis of diabetic nephropathy through reducing E2F3 expression.

Loss of lncRNA MIAT ameliorates proliferation and fibrosis of diabetic nephropathy through reducing E2F3 expression.

Journal: Journal of cellular and molecular medicine
Published: April 02, 2020

Circ-AKT3 inhibits the accumulation of extracellular matrix of mesangial cells in diabetic nephropathy via modulating miR-296-3p/E-cadherin signals.

Circ-AKT3 inhibits the accumulation of extracellular matrix of mesangial cells in diabetic nephropathy via modulating miR-296-3p/E-cadherin signals.

Journal: Journal of cellular and molecular medicine
Published: February 07, 2020

LINC00052 ameliorates acute kidney injury by sponging miR-532-3p and activating the Wnt signaling pathway.

LINC00052 ameliorates acute kidney injury by sponging miR-532-3p and activating the Wnt signaling pathway.

Journal: Aging
Published: January 05, 2020

Downregulation of XIST ameliorates acute kidney injury by sponging miR-142-5p and targeting PDCD4.

Downregulation of XIST ameliorates acute kidney injury by sponging miR-142-5p and targeting PDCD4.

Journal: Journal of cellular physiology
Published: October 25, 2019

Long non-coding RNA MEG3 promotes fibrosis and inflammatory response in diabetic nephropathy via miR-181a/Egr-1/TLR4 axis.

Long non-coding RNA MEG3 promotes fibrosis and inflammatory response in diabetic nephropathy via miR-181a/Egr-1/TLR4 axis.

Journal: Aging
Published: February 17, 2019

MicroRNA-503 contributes to podocyte injury via targeting E2F3 in diabetic nephropathy.

MicroRNA-503 contributes to podocyte injury via targeting E2F3 in diabetic nephropathy.

Journal: Journal of cellular biochemistry
Published: November 05, 2018

A simple highly sensitive and selective aptamer-based colorimetric sensor for environmental toxins microcystin-LR in water samples.

A simple highly sensitive and selective aptamer-based colorimetric sensor for environmental toxins microcystin-LR in water samples.

Journal: Journal of hazardous materials
Published: June 29, 2015

Extracellular regulated protein kinases play a key role via bone morphogenetic protein 4 in high phosphate-induced endothelial cell apoptosis.

Extracellular regulated protein kinases play a key role via bone morphogenetic protein 4 in high phosphate-induced endothelial cell apoptosis.

Journal: Life sciences
Published: October 31, 2014
Showing 1-12 of 12

Last Updated: 09/26/2026