Brian Krug
Montreal, QC H3A0C7, Canada
Last Updated: 09/26/2026
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Found 16 publications
H3K27me3 spreading organizes canonical PRC1 chromatin architecture to regulate developmental programs.
Journal: Nature genetics
Published: February 19, 2024
H3K27me3 spreading organizes canonical PRC1 chromatin architecture to regulate developmental programs.
Journal: bioRxiv : the preprint server for biology
Published: December 20, 2023
Immune landscape of oncohistone-mutant gliomas reveals diverse myeloid populations and tumor-promoting function.
Journal: Nature communications
Published: November 14, 2023
Aberrant EZHIP expression drives tumorigenesis in osteosarcoma.
Journal: Nature communications
Published: September 29, 2023
Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies.
Journal: Genome biology
Published: January 12, 2023
Author Correction: Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome.
Journal: Nature cancer
Published: June 23, 2022
Oncohistones: a roadmap to stalled development.
Journal: The FEBS journal
Published: February 17, 2021
Polycomb repressive complex 2 in the driver's seat of childhood and young adult brain tumours.
Journal: Trends in cell biology
Published: February 11, 2021
Loss of MAT2A compromises methionine metabolism and represents a vulnerability in H3K27M mutant glioma by modulating the epigenome.
Journal: Nature cancer
Published: October 16, 2020
H3K27M in Gliomas Causes a One-Step Decrease in H3K27 Methylation and Reduced Spreading within the Constraints of H3K36 Methylation.
Journal: Cell reports
Published: April 29, 2020
Last Updated: 09/26/2026