Bruno Seitaj

Leuven, VLG, Belgium

Last Updated: 09/26/2026

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Found 8 publications

Extracellular Vesicles From Mesenchymal Umbilical Cord Cells Exert Protection Against Oxidative Stress and Fibrosis in a Rat Model of Bronchopulmonary Dysplasia.

Extracellular Vesicles From Mesenchymal Umbilical Cord Cells Exert Protection Against Oxidative Stress and Fibrosis in a Rat Model of Bronchopulmonary Dysplasia.

Journal: Stem cells translational medicine

Published: November 07, 2022

TMBIM5 loss of function alters mitochondrial matrix ion homeostasis and causes a skeletal myopathy.

TMBIM5 loss of function alters mitochondrial matrix ion homeostasis and causes a skeletal myopathy.

Journal: Life science alliance

Published: April 11, 2022

Transmembrane BAX Inhibitor-1 Motif Containing Protein 5 (TMBIM5) Sustains Mitochondrial Structure, Shape, and Function by Impacting the Mitochondrial Protein Synthesis Machinery.

Transmembrane BAX Inhibitor-1 Motif Containing Protein 5 (TMBIM5) Sustains Mitochondrial Structure, Shape, and Function by Impacting the Mitochondrial Protein Synthesis Machinery.

Journal: Cells

Published: August 27, 2020

Ion and pH Sensitivity of a TMBIM Ca2+ Channel.

Ion and pH Sensitivity of a TMBIM Ca2+ Channel.

Journal: Structure (London, England : 1993)

Published: November 04, 2018

Pathophysiological consequences of isoform-specific IP3 receptor mutations.

Pathophysiological consequences of isoform-specific IP3 receptor mutations.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: May 06, 2018

Alterations in Ca2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer.

Alterations in Ca2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer.

Journal: Advances in experimental medicine and biology

Published: August 18, 2017

Modulation of Ca2+ Signaling by Anti-apoptotic B-Cell Lymphoma 2 Proteins at the Endoplasmic Reticulum-Mitochondrial Interface.

Modulation of Ca2+ Signaling by Anti-apoptotic B-Cell Lymphoma 2 Proteins at the Endoplasmic Reticulum-Mitochondrial Interface.

Journal: Frontiers in oncology

Published: January 20, 2017

The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibition.

The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibition.

Journal: Oncotarget

Published: March 03, 2016

Showing 1-8 of 8

Last Updated: 09/26/2026