Bunsyo Shiotani

Shinjuku, 1600023, Japan

Last Updated: 09/26/2026

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Found 14 publications

Tolerance of Oncogene-Induced Replication Stress: A Fuel for Genomic Instability.

Tolerance of Oncogene-Induced Replication Stress: A Fuel for Genomic Instability.

Journal: Cancers
Published: September 12, 2024

PARP inhibition-associated heterochromatin confers increased DNA replication stress and vulnerability to ATR inhibition in SMARCA4-deficient cells.

PARP inhibition-associated heterochromatin confers increased DNA replication stress and vulnerability to ATR inhibition in SMARCA4-deficient cells.

Journal: Cell death discovery
Published: August 09, 2024

An ATR-PrimPol pathway confers tolerance to oncogenic KRAS-induced and heterochromatin-associated replication stress.

An ATR-PrimPol pathway confers tolerance to oncogenic KRAS-induced and heterochromatin-associated replication stress.

Journal: Nature communications
Published: May 11, 2023

Emerging strategies for cancer therapy by ATR inhibitors.

Emerging strategies for cancer therapy by ATR inhibitors.

Journal: Cancer science
Published: March 09, 2023

p130RB2 positively contributes to ATR activation in response to replication stress via the RPA32-ETAA1 axis.

p130RB2 positively contributes to ATR activation in response to replication stress via the RPA32-ETAA1 axis.

Journal: Biochimica et biophysica acta. Molecular cell research
Published: May 05, 2022

PRPF19 regulates p53-dependent cellular senescence by modulating alternative splicing of MDM4 mRNA.

PRPF19 regulates p53-dependent cellular senescence by modulating alternative splicing of MDM4 mRNA.

Journal: The Journal of biological chemistry
Published: March 27, 2021

SMARCA4 deficiency-associated heterochromatin induces intrinsic DNA replication stress and susceptibility to ATR inhibition in lung adenocarcinoma.

SMARCA4 deficiency-associated heterochromatin induces intrinsic DNA replication stress and susceptibility to ATR inhibition in lung adenocarcinoma.

Journal: NAR cancer
Published: December 25, 2019

Correction for Matsunuma et al., "UV Damage-Induced Phosphorylation of HBO1 Triggers CRL4DDB2-Mediated Degradation To Regulate Cell Proliferation".

Correction for Matsunuma et al., "UV Damage-Induced Phosphorylation of HBO1 Triggers CRL4DDB2-Mediated Degradation To Regulate Cell Proliferation".

Journal: Molecular and cellular biology
Published: March 17, 2018

Human Rad52 Promotes XPG-Mediated R-loop Processing to Initiate Transcription-Associated Homologous Recombination Repair.

Human Rad52 Promotes XPG-Mediated R-loop Processing to Initiate Transcription-Associated Homologous Recombination Repair.

Journal: Cell
Published: December 11, 2017

Frequent amplification of receptor tyrosine kinase genes in welldifferentiated/ dedifferentiated liposarcoma.

Frequent amplification of receptor tyrosine kinase genes in welldifferentiated/ dedifferentiated liposarcoma.

Journal: Oncotarget
Published: September 26, 2016

UV Damage-Induced Phosphorylation of HBO1 Triggers CRL4DDB2-Mediated Degradation To Regulate Cell Proliferation.

UV Damage-Induced Phosphorylation of HBO1 Triggers CRL4DDB2-Mediated Degradation To Regulate Cell Proliferation.

Journal: Molecular and cellular biology
Published: August 18, 2015

CBP-93872 inhibits NBS1-mediated ATR activation, abrogating maintenance of the DNA double-strand break-specific G2 checkpoint.

CBP-93872 inhibits NBS1-mediated ATR activation, abrogating maintenance of the DNA double-strand break-specific G2 checkpoint.

Journal: Cancer research
Published: May 31, 2014
Showing 1-12 of 14

Last Updated: 09/26/2026