Caner Yildirim

Merkez Street
Kars, 36100, Turkey

Last Updated: 09/26/2026

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Found 10 publications

Naringin preserved cognitive function through modulation of cholinergic dysfunction, oxidative stress, and synaptic proteins in scopolamine-induced cognitive impairment.

Naringin preserved cognitive function through modulation of cholinergic dysfunction, oxidative stress, and synaptic proteins in scopolamine-induced cognitive impairment.

Journal: Progress in neuro-psychopharmacology & biological psychiatry

Published: May 06, 2026

Vitexin Protects Against Scopolamine-Induced Cognitive Impairment by Preserving Synaptic Integrity and Modulating Nrf2/HO-1 and NF-κB Signaling Pathways.

Vitexin Protects Against Scopolamine-Induced Cognitive Impairment by Preserving Synaptic Integrity and Modulating Nrf2/HO-1 and NF-κB Signaling Pathways.

Journal: Molecular neurobiology

Published: May 01, 2025

Fisetin mitigates cisplatin-induced peripheral neuropathy: a behavioral and immunohistochemical study.

Fisetin mitigates cisplatin-induced peripheral neuropathy: a behavioral and immunohistochemical study.

Journal: Molecular biology reports

Published: January 17, 2025

Sinapic Acid Attenuated Cisplatin-Induced Cardiotoxicity by Inhibiting Oxidative Stress and İnflammation with GPX4-Mediated NF-kB Modulation.

Sinapic Acid Attenuated Cisplatin-Induced Cardiotoxicity by Inhibiting Oxidative Stress and İnflammation with GPX4-Mediated NF-kB Modulation.

Journal: Cardiovascular toxicology

Published: August 22, 2022

Boric acid improves the behavioral, electrophysiological and histological parameters of cisplatin-induced peripheral neuropathy in rats.

Boric acid improves the behavioral, electrophysiological and histological parameters of cisplatin-induced peripheral neuropathy in rats.

Journal: Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS)

Published: September 21, 2021

KATP and TRPM2-like channels couple metabolic status to resting membrane potential of octopus neurons in the mouse ventral cochlear nucleus.

KATP and TRPM2-like channels couple metabolic status to resting membrane potential of octopus neurons in the mouse ventral cochlear nucleus.

Journal: Brain research bulletin

Published: October 23, 2020

Acid-sensing ion channels (ASICs) influence excitability of stellate neurons in the mouse cochlear nucleus.

Acid-sensing ion channels (ASICs) influence excitability of stellate neurons in the mouse cochlear nucleus.

Journal: Journal of comparative physiology. A, Neuroethology, sensory, neural, and behavioral physiology

Published: February 20, 2019

Regular exercise, overweight/obesity and sedentary lifestyle cause adaptive changes in thiol-disulfide homeostasis.

Regular exercise, overweight/obesity and sedentary lifestyle cause adaptive changes in thiol-disulfide homeostasis.

Journal: Anais da Academia Brasileira de Ciencias

Published: June 04, 2018

ERG Channels Regulate Excitability in Stellate and Bushy Cells of Mice Ventral Cochlear Nucleus.

ERG Channels Regulate Excitability in Stellate and Bushy Cells of Mice Ventral Cochlear Nucleus.

Journal: The Journal of membrane biology

Published: March 21, 2018

Modulation of Excitability of Stellate Neurons in the Ventral Cochlear Nucleus of Mice by ATP-Sensitive Potassium Channels.

Modulation of Excitability of Stellate Neurons in the Ventral Cochlear Nucleus of Mice by ATP-Sensitive Potassium Channels.

Journal: The Journal of membrane biology

Published: May 21, 2017

Showing 1-10 of 10

Last Updated: 09/26/2026