Carmen Foces-Choya

Madrid, MD, Spain

Last Updated: 09/26/2026

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Found 16 publications

MICU proteins facilitate Ca2+-dependent mitochondrial metabolon formation to regulate cellular energetics - independent of MCU.

MICU proteins facilitate Ca2+-dependent mitochondrial metabolon formation to regulate cellular energetics - independent of MCU.

Journal: Research square
Published: July 18, 2025

YAP1 and QSER1 are Key Modulators of Embryonic Signaling Pathways in the Mammalian Epiblast.

YAP1 and QSER1 are Key Modulators of Embryonic Signaling Pathways in the Mammalian Epiblast.

Journal: bioRxiv : the preprint server for biology
Published: July 16, 2025

MICU proteins facilitate calcium-dependent mitochondrial metabolon formation to regulate cellular energetics independently of MCU.

MICU proteins facilitate calcium-dependent mitochondrial metabolon formation to regulate cellular energetics independently of MCU.

Journal: Nature metabolism
Published: June 19, 2025

Integrated Systems Biology Identifies Disruptions in Mitochondrial Function and Metabolism as Key Contributors to HFpEF.

Integrated Systems Biology Identifies Disruptions in Mitochondrial Function and Metabolism as Key Contributors to HFpEF.

Journal: JACC. Basic to translational science
Published: March 18, 2025

YAP1 and QSER1 are key modulators of embryonic signaling pathways in the mammalian epiblast.

YAP1 and QSER1 are key modulators of embryonic signaling pathways in the mammalian epiblast.

Journal: EMBO reports
Published: November 03, 2024

Integrated systems biology identifies disruptions in mitochondrial function and metabolism as key contributors to heart failure with preserved ejection fraction (HFpEF).

Integrated systems biology identifies disruptions in mitochondrial function and metabolism as key contributors to heart failure with preserved ejection fraction (HFpEF).

Journal: bioRxiv : the preprint server for biology
Published: November 01, 2024

The mitochondrial Na+/Ca2+ exchanger NCLX is implied in the activation of hypoxia-inducible factors.

The mitochondrial Na+/Ca2+ exchanger NCLX is implied in the activation of hypoxia-inducible factors.

Journal: Redox biology
Published: July 15, 2024

Inhibition of ALK3-mediated signalling pathway protects against acetaminophen-induced liver injury.

Inhibition of ALK3-mediated signalling pathway protects against acetaminophen-induced liver injury.

Journal: Redox biology
Published: January 19, 2024

TMEM65 regulates NCLX-dependent mitochondrial calcium efflux.

TMEM65 regulates NCLX-dependent mitochondrial calcium efflux.

Journal: bioRxiv : the preprint server for biology
Published: October 24, 2023

TMEM65 regulates and is required for NCLX-dependent mitochondrial calcium efflux.

TMEM65 regulates and is required for NCLX-dependent mitochondrial calcium efflux.

Journal: Nature metabolism
Published: October 16, 2023

Corrigendum to "Downregulation of Thioredoxin-1-dependent CD95 S-nitrosation by Sorafenib reduces liver cancer" [Redox Biol. 34 (2020) 101528].

Corrigendum to "Downregulation of Thioredoxin-1-dependent CD95 S-nitrosation by Sorafenib reduces liver cancer" [Redox Biol. 34 (2020) 101528].

Journal: Redox biology
Published: July 21, 2023

Cigarette smoke induces pulmonary arterial dysfunction through an imbalance in the redox status of the soluble guanylyl cyclase.

Cigarette smoke induces pulmonary arterial dysfunction through an imbalance in the redox status of the soluble guanylyl cyclase.

Journal: Free radical biology & medicine
Published: March 31, 2022
Showing 1-12 of 16

Last Updated: 09/26/2026