Catherine A. Blizzard

17 Liverpool Street
Hobart, TAS 7000, Australia

Last Updated: 09/26/2026

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Found 34 publications

Amyotrophic lateral sclerosis: A rare but aggressive adult-onset disease.

Amyotrophic lateral sclerosis: A rare but aggressive adult-onset disease.

Journal: Trends in molecular medicine

Published: March 01, 2026

Modeling amyotrophic lateral sclerosis (ALS) in vitro: from mechanistic studies to translatable drug discovery.

Modeling amyotrophic lateral sclerosis (ALS) in vitro: from mechanistic studies to translatable drug discovery.

Journal: Lab on a chip

Published: January 07, 2026

Synaptic changes contribute to persistent extra-motor behaviour deficits in amyotrophic lateral sclerosis.

Synaptic changes contribute to persistent extra-motor behaviour deficits in amyotrophic lateral sclerosis.

Journal: Acta neuropathologica communications

Published: May 18, 2025

Demyelination Produces a Shift in the Population of Cortical Neurons That Synapse with Callosal Oligodendrocyte Progenitor Cells.

Demyelination Produces a Shift in the Population of Cortical Neurons That Synapse with Callosal Oligodendrocyte Progenitor Cells.

Journal: eNeuro

Published: March 10, 2025

Synaptic remodeling follows upper motor neuron hyperexcitability in a rodent model of TDP-43.

Synaptic remodeling follows upper motor neuron hyperexcitability in a rodent model of TDP-43.

Journal: Frontiers in cellular neuroscience

Published: August 09, 2023

Intranasal neuropeptide Y1 receptor antagonism improves motor deficits in symptomatic SOD1 ALS mice.

Intranasal neuropeptide Y1 receptor antagonism improves motor deficits in symptomatic SOD1 ALS mice.

Journal: Annals of clinical and translational neurology

Published: July 31, 2023

How do we get from hyperexcitability to excitotoxicity in amyotrophic lateral sclerosis?

How do we get from hyperexcitability to excitotoxicity in amyotrophic lateral sclerosis?

Journal: Brain : a journal of neurology

Published: July 17, 2023

Pathologically mislocalised TDP-43 in upper motor neurons causes a die-forward spread of ALS-like pathogenic changes throughout the mouse corticomotor system.

Pathologically mislocalised TDP-43 in upper motor neurons causes a die-forward spread of ALS-like pathogenic changes throughout the mouse corticomotor system.

Journal: Progress in neurobiology

Published: September 08, 2022

Estrogen Enhances Dendrite Spine Function and Recovers Deficits in Neuroplasticity in the prpTDP-43A315T Mouse Model of Amyotrophic Lateral Sclerosis.

Estrogen Enhances Dendrite Spine Function and Recovers Deficits in Neuroplasticity in the prpTDP-43A315T Mouse Model of Amyotrophic Lateral Sclerosis.

Journal: Molecular neurobiology

Published: September 05, 2021

Cytoplasmic Human TDP-43 Mislocalization Induces Widespread Dendritic Spine Loss in Mouse Upper Motor Neurons.

Cytoplasmic Human TDP-43 Mislocalization Induces Widespread Dendritic Spine Loss in Mouse Upper Motor Neurons.

Journal: Brain sciences

Published: May 31, 2021

Riluzole does not ameliorate disease caused by cytoplasmic TDP-43 in a mouse model of amyotrophic lateral sclerosis.

Riluzole does not ameliorate disease caused by cytoplasmic TDP-43 in a mouse model of amyotrophic lateral sclerosis.

Journal: The European journal of neuroscience

Published: December 16, 2020

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

Efficacy of epothilones in central nervous system trauma treatment: what has age got to do with it?

Journal: Neural regeneration research

Published: October 16, 2020

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Last Updated: 09/26/2026