Changhua Qu

No.169, Donghu Road
Wuhan, 430071, China

Last Updated: 09/26/2026

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Found 13 publications

FTO Relieves Oxygen-glucose Deprivation/reoxygenation-induced Cerebral Ischemia-reperfusion Injury through the m6A Demethylation of Gpr84 mRNA.

FTO Relieves Oxygen-glucose Deprivation/reoxygenation-induced Cerebral Ischemia-reperfusion Injury through the m6A Demethylation of Gpr84 mRNA.

Journal: Shock (Augusta, Ga.)

Published: February 24, 2026

Exogenous branched chain amino acids improve cognitive impairment by regulating glutamatergic synapses in chronic cerebral hypoperfusion rats.

Exogenous branched chain amino acids improve cognitive impairment by regulating glutamatergic synapses in chronic cerebral hypoperfusion rats.

Journal: Nutritional neuroscience

Published: July 30, 2025

White matter hyperintensities mediate the association between frailty and cognitive impairment in moyamoya disease.

White matter hyperintensities mediate the association between frailty and cognitive impairment in moyamoya disease.

Journal: Clinical neurology and neurosurgery

Published: December 09, 2023

The environmental enrichment ameliorates chronic cerebral hypoperfusion-induced cognitive impairment by activating autophagy signaling pathway and improving synaptic function in hippocampus.

The environmental enrichment ameliorates chronic cerebral hypoperfusion-induced cognitive impairment by activating autophagy signaling pathway and improving synaptic function in hippocampus.

Journal: Brain research bulletin

Published: February 11, 2023

The environmental enrichment ameliorates chronic unpredictable mild stress-induced depressive-like behaviors and cognitive decline by inducing autophagy-mediated inflammation inhibition.

The environmental enrichment ameliorates chronic unpredictable mild stress-induced depressive-like behaviors and cognitive decline by inducing autophagy-mediated inflammation inhibition.

Journal: Brain research bulletin

Published: March 30, 2022

Enriched environment priors to TET1 hippocampal administration for regulating psychiatric behaviors via glial reactivity in chronic cerebral hypoperfusion models.

Enriched environment priors to TET1 hippocampal administration for regulating psychiatric behaviors via glial reactivity in chronic cerebral hypoperfusion models.

Journal: Journal of affective disorders

Published: January 23, 2022

Dimethyl fumarate improves cognitive deficits in chronic cerebral hypoperfusion rats by alleviating inflammation, oxidative stress, and ferroptosis via NRF2/ARE/NF-κB signal pathway.

Dimethyl fumarate improves cognitive deficits in chronic cerebral hypoperfusion rats by alleviating inflammation, oxidative stress, and ferroptosis via NRF2/ARE/NF-κB signal pathway.

Journal: International immunopharmacology

Published: February 11, 2021

A High Resolution MRI Study of the Relationship Between Plaque Enhancement and Perforator Stroke after Stenting for Symptomatic Vertebrobasilar Artery Stenosis.

A High Resolution MRI Study of the Relationship Between Plaque Enhancement and Perforator Stroke after Stenting for Symptomatic Vertebrobasilar Artery Stenosis.

Journal: Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association

Published: October 10, 2020

Improvement of autophagy dysfunction as a potential mechanism for environmental enrichment to protect blood-brain barrier in rats with vascular cognitive impairment.

Improvement of autophagy dysfunction as a potential mechanism for environmental enrichment to protect blood-brain barrier in rats with vascular cognitive impairment.

Journal: Neuroscience letters

Published: October 06, 2020

Nuclear receptor TLX may be through regulating the SIRT1/NF-κB pathway to ameliorate cognitive impairment in chronic cerebral hypoperfusion.

Nuclear receptor TLX may be through regulating the SIRT1/NF-κB pathway to ameliorate cognitive impairment in chronic cerebral hypoperfusion.

Journal: Brain research bulletin

Published: May 25, 2020

Mfsd2a Reverses Spatial Learning and Memory Impairment Caused by Chronic Cerebral Hypoperfusion via Protection of the Blood-Brain Barrier.

Mfsd2a Reverses Spatial Learning and Memory Impairment Caused by Chronic Cerebral Hypoperfusion via Protection of the Blood-Brain Barrier.

Journal: Frontiers in neuroscience

Published: December 27, 2019

Protection of blood-brain barrier as a potential mechanism for enriched environments to improve cognitive impairment caused by chronic cerebral hypoperfusion.

Protection of blood-brain barrier as a potential mechanism for enriched environments to improve cognitive impairment caused by chronic cerebral hypoperfusion.

Journal: Behavioural brain research

Published: June 29, 2019

Showing 1-12 of 13

Last Updated: 09/26/2026