Charles S. Redwood

Oxford, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 55 publications

Structural, biophysical and cellular assessment of filamin C M82K: A test case for VUS interpretation in cardiomyopathy.

Structural, biophysical and cellular assessment of filamin C M82K: A test case for VUS interpretation in cardiomyopathy.

Journal: Journal of molecular and cellular cardiology

Published: December 19, 2025

Structural, biophysical and cellular assessment of filamin C M82K: A test case for VUS interpretation in cardiomyopathy.

Structural, biophysical and cellular assessment of filamin C M82K: A test case for VUS interpretation in cardiomyopathy.

Journal: Journal of molecular and cellular cardiology

Published: December 19, 2025

Comprehensive biophysical and structural profiling of alpha-actinin-2 variants reveals mechanistic diversity in hypertrophic cardiomyopathy.

Comprehensive biophysical and structural profiling of alpha-actinin-2 variants reveals mechanistic diversity in hypertrophic cardiomyopathy.

Journal: Nature communications

Published: November 18, 2025

Regulatory T cells attenuate chronic inflammation and cardiac fibrosis in hypertrophic cardiomyopathy.

Regulatory T cells attenuate chronic inflammation and cardiac fibrosis in hypertrophic cardiomyopathy.

Journal: Science translational medicine

Published: July 09, 2025

An ALPK3 truncation variant causing autosomal dominant hypertrophic cardiomyopathy is partially rescued by mavacamten.

An ALPK3 truncation variant causing autosomal dominant hypertrophic cardiomyopathy is partially rescued by mavacamten.

Journal: Scientific reports

Published: October 09, 2024

Nutraceuticals silybin B, resveratrol, and epigallocatechin-3 gallate-bind to cardiac muscle troponin to restore the loss of lusitropy caused by cardiomyopathy mutations in vitro, in vivo, and in silico.

Nutraceuticals silybin B, resveratrol, and epigallocatechin-3 gallate-bind to cardiac muscle troponin to restore the loss of lusitropy caused by cardiomyopathy mutations in vitro, in vivo, and in silico.

Journal: Frontiers in physiology

Published: September 01, 2024

Molecular Mechanisms of Deregulation of Muscle Contractility Caused by the R168H Mutation in TPM3 and Its Attenuation by Therapeutic Agents.

Molecular Mechanisms of Deregulation of Muscle Contractility Caused by the R168H Mutation in TPM3 and Its Attenuation by Therapeutic Agents.

Journal: International journal of molecular sciences

Published: February 28, 2023

Comparing the effects of chemical Ca2+ dyes and R-GECO on contractility and Ca2+ transients in adult and human iPSC cardiomyocytes.

Comparing the effects of chemical Ca2+ dyes and R-GECO on contractility and Ca2+ transients in adult and human iPSC cardiomyocytes.

Journal: Journal of molecular and cellular cardiology

Published: November 25, 2022

Disease modeling of a mutation in α-actinin 2 guides clinical therapy in hypertrophic cardiomyopathy.

Disease modeling of a mutation in α-actinin 2 guides clinical therapy in hypertrophic cardiomyopathy.

Journal: EMBO molecular medicine

Published: June 08, 2022

Low expression of the K280N TNNT2 mutation is sufficient to increase basal myofilament activation in human hypertrophy cardiomyopathy.

Low expression of the K280N TNNT2 mutation is sufficient to increase basal myofilament activation in human hypertrophy cardiomyopathy.

Journal: Journal of molecular and cellular cardiology plus

Published: March 31, 2022

Genetics Matters: Voyaging from the Past into the Future of Humanity and Sustainability.

Genetics Matters: Voyaging from the Past into the Future of Humanity and Sustainability.

Journal: International journal of molecular sciences

Published: February 24, 2022

CalTrack: High-Throughput Automated Calcium Transient Analysis in Cardiomyocytes.

CalTrack: High-Throughput Automated Calcium Transient Analysis in Cardiomyocytes.

Journal: Circulation research

Published: May 21, 2021

Showing 1-12 of 55

Last Updated: 09/26/2026