Christina Golz

Mainz, RP, Germany

Last Updated: 09/26/2026

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Found 14 publications

Protease-activated receptor 4 deficiency increases mortality, intracranial bleeding, and blood-brain barrier impairment following traumatic brain injury in mice.

Protease-activated receptor 4 deficiency increases mortality, intracranial bleeding, and blood-brain barrier impairment following traumatic brain injury in mice.

Journal: Research and practice in thrombosis and haemostasis

Published: April 29, 2025

Sortilin is dispensable for secondary injury processes following traumatic brain injury in mice.

Sortilin is dispensable for secondary injury processes following traumatic brain injury in mice.

Journal: Heliyon

Published: December 22, 2023

Early DNase-I therapy delays secondary brain damage after traumatic brain injury in adult mice.

Early DNase-I therapy delays secondary brain damage after traumatic brain injury in adult mice.

Journal: Scientific reports

Published: October 26, 2022

Ribonuclease-1 treatment after traumatic brain injury preserves blood-brain barrier integrity and delays secondary brain damage in mice.

Ribonuclease-1 treatment after traumatic brain injury preserves blood-brain barrier integrity and delays secondary brain damage in mice.

Journal: Scientific reports

Published: October 05, 2021

Single intracerebroventricular progranulin injection adversely affects the blood-brain barrier in experimental traumatic brain injury.

Single intracerebroventricular progranulin injection adversely affects the blood-brain barrier in experimental traumatic brain injury.

Journal: Journal of neurochemistry

Published: February 13, 2021

Pharmacologic Inhibition of ADAM10 Attenuates Brain Tissue Loss, Axonal Injury and Pro-inflammatory Gene Expression Following Traumatic Brain Injury in Mice.

Pharmacologic Inhibition of ADAM10 Attenuates Brain Tissue Loss, Axonal Injury and Pro-inflammatory Gene Expression Following Traumatic Brain Injury in Mice.

Journal: Frontiers in cell and developmental biology

Published: January 30, 2021

Administration of all-trans retinoic acid after experimental traumatic brain injury is brain protective.

Administration of all-trans retinoic acid after experimental traumatic brain injury is brain protective.

Journal: British journal of pharmacology

Published: September 16, 2019

Sex hormones modulate pathogenic processes in experimental traumatic brain injury.

Sex hormones modulate pathogenic processes in experimental traumatic brain injury.

Journal: Journal of neurochemistry

Published: October 23, 2018

Plasminogen activator inhibitor-1 augments damage by impairing fibrinolysis after traumatic brain injury.

Plasminogen activator inhibitor-1 augments damage by impairing fibrinolysis after traumatic brain injury.

Journal: Annals of neurology

Published: July 30, 2018

RS1 (Rsc1A1) deficiency limits cerebral SGLT1 expression and delays brain damage after experimental traumatic brain injury.

RS1 (Rsc1A1) deficiency limits cerebral SGLT1 expression and delays brain damage after experimental traumatic brain injury.

Journal: Journal of neurochemistry

Published: April 05, 2018

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Journal: Frontiers in neuroscience

Published: September 07, 2017

Multifaceted Mechanisms of WY-14643 to Stabilize the Blood-Brain Barrier in a Model of Traumatic Brain Injury.

Multifaceted Mechanisms of WY-14643 to Stabilize the Blood-Brain Barrier in a Model of Traumatic Brain Injury.

Journal: Frontiers in molecular neuroscience

Published: February 09, 2017

Showing 1-12 of 14

Last Updated: 09/26/2026