Christopher I. Slape

Brisbane, QLD 4102, Australia

Last Updated: 09/26/2026

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Found 11 publications

Cell-Extrinsic Differentiation Block Mediated by EphA3 in Pre-Leukaemic Thymus Contributes to Disease Progression.

Cell-Extrinsic Differentiation Block Mediated by EphA3 in Pre-Leukaemic Thymus Contributes to Disease Progression.

Journal: Cancers
Published: May 31, 2021

Pre-Leukemic States: United by Difference.

Pre-Leukemic States: United by Difference.

Journal: Cancers
Published: March 15, 2021

Seeking commonalities in fusion proteins of NUP98.

Seeking commonalities in fusion proteins of NUP98.

Journal: Nature structural & molecular biology
Published: January 22, 2021

The NUP98-HOXD13 fusion oncogene induces thymocyte self-renewal via Lmo2/Lyl1.

The NUP98-HOXD13 fusion oncogene induces thymocyte self-renewal via Lmo2/Lyl1.

Journal: Leukemia
Published: July 16, 2018

PUMA promotes apoptosis of hematopoietic progenitors driving leukemic progression in a mouse model of myelodysplasia.

PUMA promotes apoptosis of hematopoietic progenitors driving leukemic progression in a mouse model of myelodysplasia.

Journal: Cell death and differentiation
Published: April 17, 2015

EphA3 biology and cancer.

EphA3 biology and cancer.

Journal: Growth factors (Chur, Switzerland)
Published: November 14, 2014

The fusion partner specifies the oncogenic potential of NUP98 fusion proteins.

The fusion partner specifies the oncogenic potential of NUP98 fusion proteins.

Journal: Leukemia research
Published: March 18, 2013

Inhibition of apoptosis by BCL2 prevents leukemic transformation of a murine myelodysplastic syndrome.

Inhibition of apoptosis by BCL2 prevents leukemic transformation of a murine myelodysplastic syndrome.

Journal: Blood
Published: August 03, 2012

Gene expression profiling and candidate gene resequencing identifies pathways and mutations important for malignant transformation caused by leukemogenic fusion genes.

Gene expression profiling and candidate gene resequencing identifies pathways and mutations important for malignant transformation caused by leukemogenic fusion genes.

Journal: Experimental hematology
Published: July 30, 2012

Knock-in of a FLT3/ITD mutation cooperates with a NUP98-HOXD13 fusion to generate acute myeloid leukemia in a mouse model.

Knock-in of a FLT3/ITD mutation cooperates with a NUP98-HOXD13 fusion to generate acute myeloid leukemia in a mouse model.

Journal: Blood
Published: February 11, 2012

NUP98 gene fusions and hematopoietic malignancies: common themes and new biologic insights.

NUP98 gene fusions and hematopoietic malignancies: common themes and new biologic insights.

Journal: Blood
Published: September 28, 2011
Showing 1-11 of 11

Last Updated: 09/26/2026