Christopher M. Tay

Melbourne, VIC, Australia

Last Updated: 09/26/2026

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Found 11 publications

B Cell and CD4 T Cell Interactions Promote Development of Atherosclerosis.

B Cell and CD4 T Cell Interactions Promote Development of Atherosclerosis.

Journal: Frontiers in immunology

Published: October 11, 2019

Anti-TIM-1 Monoclonal Antibody (RMT1-10) Attenuates Atherosclerosis By Expanding IgM-producing B1a Cells.

Anti-TIM-1 Monoclonal Antibody (RMT1-10) Attenuates Atherosclerosis By Expanding IgM-producing B1a Cells.

Journal: Journal of the American Heart Association

Published: June 25, 2018

Follicular B Cells Promote Atherosclerosis via T Cell-Mediated Differentiation Into Plasma Cells and Secreting Pathogenic Immunoglobulin G.

Follicular B Cells Promote Atherosclerosis via T Cell-Mediated Differentiation Into Plasma Cells and Secreting Pathogenic Immunoglobulin G.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: July 27, 2017

Toll-Like Receptor (TLR)4 and MyD88 are Essential for Atheroprotection by Peritoneal B1a B Cells.

Toll-Like Receptor (TLR)4 and MyD88 are Essential for Atheroprotection by Peritoneal B1a B Cells.

Journal: Journal of the American Heart Association

Published: December 09, 2016

B-cell-specific depletion of tumour necrosis factor alpha inhibits atherosclerosis development and plaque vulnerability to rupture by reducing cell death and inflammation.

B-cell-specific depletion of tumour necrosis factor alpha inhibits atherosclerosis development and plaque vulnerability to rupture by reducing cell death and inflammation.

Journal: Cardiovascular research

Published: May 03, 2016

Phosphatidylserine liposomes mimic apoptotic cells to attenuate atherosclerosis by expanding polyreactive IgM producing B1a lymphocytes.

Phosphatidylserine liposomes mimic apoptotic cells to attenuate atherosclerosis by expanding polyreactive IgM producing B1a lymphocytes.

Journal: Cardiovascular research

Published: December 22, 2014

Protective role for Toll-like receptor-9 in the development of atherosclerosis in apolipoprotein E-deficient mice.

Protective role for Toll-like receptor-9 in the development of atherosclerosis in apolipoprotein E-deficient mice.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: January 18, 2014

Oral health and dental treatment needs of people with motor neurone disease.

Oral health and dental treatment needs of people with motor neurone disease.

Journal: Australian dental journal

Published: December 02, 2013

Cytotoxic and proinflammatory CD8+ T lymphocytes promote development of vulnerable atherosclerotic plaques in apoE-deficient mice.

Cytotoxic and proinflammatory CD8+ T lymphocytes promote development of vulnerable atherosclerotic plaques in apoE-deficient mice.

Journal: Circulation

Published: February 12, 2013

BAFF receptor mAb treatment ameliorates development and progression of atherosclerosis in hyperlipidemic ApoE(-/-) mice.

BAFF receptor mAb treatment ameliorates development and progression of atherosclerosis in hyperlipidemic ApoE(-/-) mice.

Journal: PloS one

Published: February 05, 2013

Depletion of B2 but not B1a B cells in BAFF receptor-deficient ApoE mice attenuates atherosclerosis by potently ameliorating arterial inflammation.

Depletion of B2 but not B1a B cells in BAFF receptor-deficient ApoE mice attenuates atherosclerosis by potently ameliorating arterial inflammation.

Journal: PloS one

Published: October 12, 2011

Showing 1-11 of 11

Last Updated: 09/26/2026