Chun-chun Liu

Nanjing, China

Last Updated: 09/26/2026

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Found 16 publications

Two novel compounds inhibit Flavivirus infection in vitro and in vivo by targeting lipid metabolism.

Two novel compounds inhibit Flavivirus infection in vitro and in vivo by targeting lipid metabolism.

Journal: Journal of virology
Published: August 19, 2024

The Small GTPase Rab14 Regulates the Trafficking of Ceramide from Endoplasmic Reticulum to Golgi Apparatus and Facilitates Classical Swine Fever Virus Assembly.

The Small GTPase Rab14 Regulates the Trafficking of Ceramide from Endoplasmic Reticulum to Golgi Apparatus and Facilitates Classical Swine Fever Virus Assembly.

Journal: Journal of virology
Published: May 31, 2023

Intracellular Vimentin Regulates the Formation of Classical Swine Fever Virus Replication Complex through Interaction with NS5A Protein.

Intracellular Vimentin Regulates the Formation of Classical Swine Fever Virus Replication Complex through Interaction with NS5A Protein.

Journal: Journal of virology
Published: May 02, 2023

Kinesin-1 Regulates Endocytic Trafficking of Classical Swine Fever Virus along Acetylated Microtubules.

Kinesin-1 Regulates Endocytic Trafficking of Classical Swine Fever Virus along Acetylated Microtubules.

Journal: Journal of virology
Published: January 05, 2023

Corrigendum to "Valosin-containing protein (VCP/p97) is responsible for the endocytotic trafficking of classical swine fever virus" [Vet. Microbiol. 272 (2022) 109511].

Corrigendum to "Valosin-containing protein (VCP/p97) is responsible for the endocytotic trafficking of classical swine fever virus" [Vet. Microbiol. 272 (2022) 109511].

Journal: Veterinary microbiology
Published: September 10, 2022

Valosin-containing protein (VCP/p97) is responsible for the endocytotic trafficking of classical swine fever virus.

Valosin-containing protein (VCP/p97) is responsible for the endocytotic trafficking of classical swine fever virus.

Journal: Veterinary microbiology
Published: May 10, 2022

Cellular ESCRT components are recruited to regulate the endocytic trafficking and RNA replication compartment assembly during classical swine fever virus infection.

Cellular ESCRT components are recruited to regulate the endocytic trafficking and RNA replication compartment assembly during classical swine fever virus infection.

Journal: PLoS pathogens
Published: November 04, 2021

Fatty Acid Synthase Is Involved in Classical Swine Fever Virus Replication by Interaction with NS4B.

Fatty Acid Synthase Is Involved in Classical Swine Fever Virus Replication by Interaction with NS4B.

Journal: Journal of virology
Published: June 16, 2021

Microfilaments and microtubules alternately coordinate the multi-step endosomal trafficking of Classical Swine Fever Virus.

Microfilaments and microtubules alternately coordinate the multi-step endosomal trafficking of Classical Swine Fever Virus.

Journal: Journal of virology
Published: February 25, 2021

Curcumin inhibits classical swine fever virus replication by interfering with lipid metabolism.

Curcumin inhibits classical swine fever virus replication by interfering with lipid metabolism.

Journal: Veterinary microbiology
Published: February 16, 2021

The ESCRT-I Subunit Tsg101 Plays Novel Dual Roles in Entry and Replication of Classical Swine Fever Virus.

The ESCRT-I Subunit Tsg101 Plays Novel Dual Roles in Entry and Replication of Classical Swine Fever Virus.

Journal: Journal of virology
Published: September 29, 2020

U18666A inhibits classical swine fever virus replication through interference with intracellular cholesterol trafficking.

U18666A inhibits classical swine fever virus replication through interference with intracellular cholesterol trafficking.

Journal: Veterinary microbiology
Published: July 02, 2019
Showing 1-12 of 16

Last Updated: 09/26/2026