Chun Z. Jin
Jilin, China
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 8 publications
Neuronal nitric oxide synthase modulation of intracellular Ca2+ handling overrides fatty acid potentiation of cardiac inotropy in hypertensive rats.
Journal: Pflugers Archiv : European journal of physiology
Published: March 23, 2017
S-nitrosylation of transglutaminase 2 impairs fatty acid-stimulated contraction in hypertensive cardiomyocytes.
Journal: Experimental & molecular medicine
Published: January 10, 2017
Modulation of L-type Ca²⁺ channel activity by neuronal nitric oxide synthase and myofilament Ca²⁺ sensitivity in cardiac myocytes from hypertensive rat.
Journal: Cell calcium
Published: January 16, 2015
ROS and endothelial nitric oxide synthase (eNOS)-dependent trafficking of angiotensin II type 2 receptor begets neuronal NOS in cardiac myocytes.
Journal: Basic research in cardiology
Published: December 31, 2014
Molecular mechanisms of neuronal nitric oxide synthase in cardiac function and pathophysiology.
Journal: The Journal of physiology
Published: April 24, 2014
Low K⁺ current in arterial myocytes with impaired K⁺-vasodilation and its recovery by exercise in hypertensive rats.
Journal: Pflugers Archiv : European journal of physiology
Published: December 15, 2013
Myofilament Ca2+ desensitization mediates positive lusitropic effect of neuronal nitric oxide synthase in left ventricular myocytes from murine hypertensive heart.
Journal: Journal of molecular and cellular cardiology
Published: October 12, 2012
Neuronal nitric oxide synthase is up-regulated by angiotensin II and attenuates NADPH oxidase activity and facilitates relaxation in murine left ventricular myocytes.
Journal: Journal of molecular and cellular cardiology
Published: December 08, 2011
Showing 1-8 of 8
Last Updated: 09/26/2026